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Archive for the ‘Longevity’ Category

life span | Britannica.com

Thursday, August 4th, 2016

Alternative title: longevity

Life span, mayflyG.E. HydeNHPA/EB Inc.the period of time between the birth and death of an organism.

longevity: animalsEncyclopdia Britannica, Inc.It is a commonplace that all organisms die. Some die after only a brief existence, like that of the mayfly, whose adult life burns out in a day, and others like that of the gnarled bristlecone pines, which have lived thousands of years. The limits of the life span of each species appear to be determined ultimately by heredity. Locked within the code of the genetic material are instructions that specify the age beyond which a species cannot live given even the most favourable conditions. And many environmental factors act to diminish that upper age limit.

The maximum life span is a theoretical number whose exact value cannot be determined from existing knowledge about an organism; it is often given as a rough estimate based on the longest lived organism of its species known to date. A more meaningful measure is the average life span; this is a statistical concept that is derived by the analysis of mortality data for populations of each species. A related term is the expectation of life, a hypothetical number computed for humans from mortality tables drawn up by insurance companies. Life expectancy represents the average number of years that a group of persons, all born at the same time, might be expected to live, and it is based on the changing death rate over many past years.

The concept of life span implies that there is an individual whose existence has a definite beginning and end. What constitutes the individual in most cases presents no problem: among organisms that reproduce sexually the individual is a certain amount of living substance capable of maintaining itself alive and endowed with hereditary features that are in some measure unique. In some organisms, however, extensive and apparently indefinite growth takes place and reproduction may occur by division of a single parent organism, as in many protists, including bacteria, algae, and protozoans. If these divisions are incomplete, a colony results; if the parts separate, genetically identical organisms are formed. In order to consider life span in such organisms, the individual must be defined arbitrarily since the organisms are continually dividing. In a strict sense, the life spans in such instances are not comparable to those forms that are sexually produced.

The beginning of an organism can be defined by the formation of the fertilized egg in sexual forms; or by the physical separation of the new organism in asexual forms (many invertebrate animals and many plants). In animals generally, birth is considered to be the beginning of the life span. The timing of birth, however, is so different in various animals that it is only a poor criterion. In many marine invertebrates the hatchling larva consists of relatively few cells, not nearly so far along toward adulthood as a newborn mammal. For even among mammals, variations are considerable. A kangaroo at birth is about an inch long and must develop further in the pouch, hardly comparable to a newborn deer, who within minutes is walking about. If life spans of different kinds of organisms are to be compared, it is essential that these variations be accounted for. The end of an organisms existence results when irreversible changes have occurred to such an extent that the individual no longer actively retains its organization. There is thus a brief period during which it is impossible to say whether the organism is still alive, but this time is so short relative to the total length of life that it creates no great problem in determining life span.

Some organisms seem to be potentially immortal. Unless an accident puts an end to life, they appear to be fully capable of surviving indefinitely. This faculty has been attributed to certain fishes and reptiles, which appear to be capable of unlimited growth. Without examining the various causes of death in detail (see death) a distinction can be made between death as a result of internal changes (i.e., aging) and death as a result of some purely external factor, such as an accident. It is notable that the absence of aging processes is correlated with the absence of individuality. In other words, organisms in which the individual is difficult to define, as in colonial forms, appear not to age.

Plants grow old as surely as do animals. However, a generally accepted definition of age in plants has not yet been realized. If the age of an individual plant is that time interval between the reproductive process that gave rise to the individual and the death of the individual, the age attained may be given readily for some kinds of plants but not for others. The Table lists maximum ages, both estimated and verified, for some seed plants.

Maximum ages for some seed plants

An English oak that has 1,000 annual rings in the trunk is 1,000 years old. But age is less certain in the case of an arctic lupine that germinated from a seed that, containing the embryo, had been lying in a lemmings burrow in the arctic permafrost for 10,000 years.

The mushroom caps that appear overnight last for only a few days, but the network of fungus filaments in the soil (the mycelia) may be as old as 400 years. Because of important differences in structure, the life span of higher plants cannot be compared with that of higher animals. Normally, embryonic cells (that is, cells capable of changing in form or becoming specialized) cease to exist very early in the life of an animal. In plants, however, embryonic tissuethe plant meristemsmay contribute to growth and tissue formation for a much longer time, in some cases throughout the life of the plant. Thus the oldest known trees, bristlecone pines of California and Nevada, have one meristem (the cambium) that has been adding cells to the diameter of these trees for, in many cases, more than 4,000 years and another meristem (the apical) that has been adding cells to the length of these trees for the same period. These meristematic tissues are as old as the plant itself; they were formed in the embryo. The wood, bark, leaves and cones, however, live for only a few years. The wood of the trunk and roots, although dead, remains a part of the tree indefinitely, but the bark, leaves, and cones are continually in the process of dying and sloughing off.

Among the lower plants only a few mosses possess structures that enable an estimate of their age to be made. The haircap moss (Polytrichum) grows through its own stem tip each year, leaving a ring of scales that marks the annual growth. Three to five years growth in this moss is common, but life spans of 10 years have been recorded. The lower portions of such a moss are dead, though intact. Peat moss (Sphagnum) forms extensive growths that fill acid bogs with a peaty turf consisting of the dead lower portions of mosses whose living tops continue growing. Mosses that become encrusted with lime (calcium carbonate) and form tufa beds several metres thick also have living tips and dead lower portions. On the basis of their observed annual growth, some tufa mosses are estimated to have been growing for as long as 2,800 years.

No reliable method for determining the age of ferns exists, but on the basis of size attained and growth rate, some tree ferns are thought to be several decades old. Some club mosses, or lycopsids, have a storied growth pattern similar to that of the haircap moss. Under favourable conditions some specimens live five to seven years.

The woody seed plants, such as conifers and broadleaf trees, are the most amenable to determination of age. In temperate regions, where each years growth is brought to an end by cold or dryness, every growth period is limited by an annual ringa new layer of wood added to the diameter of the tree. These rings may be counted on the cut ends of a tree that has been felled or, using a special instrument, a cylinder of wood can be cut out and the growth rings counted and studied. In the far north growth rings are so close together that they are difficult to count. In the moist tropics growth is more or less continuous, so that clearly defined rings are difficult to find.

Often the age of a tree is estimated on the basis of its diameter, especially when the average annual increase in diameter is known. The source of greatest error in this method is the not infrequent fusing of the trunks of more than one tree, as, for example, occurred in a Montezuma cypress in Santa Mara del Tule, a little Mexican village near Oaxaca. This tree, described by the Spanish explorer Hernan Corts in the early 1500s, was earlier estimated on the basis of its great thickness to be 6,000 years old; later studies, however, proved it to be three trees grown together. Estimates of the age of some English yews have been as high as 3,000 years, but these figures, too, have turned out to be based on the fusion of close-growing trunks, none of which is more than 250 years old. Increment borings of bristlecone pines have shown specimens in the western United States to be 4,600 years old.

Plants, usually herbaceous, that live for only one growing season and produce flowers and seeds in that time are called annuals. They may be represented by such plants as corn and marigolds, which spend a period of a few weeks to a few months rapidly accumulating food materials. As a result of hormonal changesbrought about in many plants by changes in environmental factors such as day length and temperatureleaf-producing tissues change abruptly to flower-producing ones. The formation of flowers, fruits, and seeds rapidly depletes food reserves and the vegetative portion of the plant usually dies. Although the exhaustion of food reserves often accompanies death of the plant, it is not necessarily the cause of death.

These plants, too, are usually herbaceous. They live for two growing seasons. During the first season, food is accumulated, usually in a thickened root (beets, carrots); flowering occurs in the second season. As in annuals, flowering exhausts the food reserves, and the plants die after the seeds mature.

These plants have a life span of several to many years. Some are herbaceous (iris, delphinium), others are shrubs or trees. The perennials differ from the above-mentioned groups in that the storage structures are either permanent or are renewed each year. Perennials require from one to many years growth before flowering. The preflowering (juvenile) period is usually shorter in trees and shrubs with shorter life spans than in those with longer life spans. The long-lived beech tree (Fagus sylvatica), for example, passes 3040 years in the juvenile stage, during which time there is rapid growth but no flowering.

Some plantscotton and tomatoes, for exampleare perennials in their native tropical regions but are capable of blooming and producing fruits, seeds, or other useful parts in their first year. Such plants are often grown as annuals in the temperate zones.

Although there is great variety in the longevity of seeds, the dormant embryo plant contained within the seed will lose its viability (ability to grow) if germination fails to occur within a certain time. Reports of the sprouting of wheat taken from Egyptian tombs are unfounded, but some seeds do retain their viability a long time. Indian lotus seeds (actually fruits) have the longest known retention of viability. On the other hand, seeds of some willows lose their ability to germinate within a week after they have reached maturity.

The loss of viability of seeds in storage, although hastened or retarded by environmental factors, is the result of changes that take place within the seed itself. The changes that have been investigated are: exhaustion of food supply; gradual denaturing or loss of vital structure by protoplasmic proteins; breakdown of enzymes; accumulation of toxins resulting from the metabolism of the seed. Some self-produced toxins may cause mutations that hamper seed germination. Since seeds of different species vary greatly in structure, physiology, and life history, no single set of age factors can apply to all seeds.

Much of what is known of the length of life of animals other than man derives from observations of domesticated species in laboratories and zoos. One has only to consider how few animals reveal their age to appreciate the difficulties involved in answering the apparently simple question of how long they live in nature. In many fishes, a few kinds of clams, and an occasional species of other groups, growth is seasonal, so that annual zones of growth, much like tree rings, are produced in some part of the organism. Among game species, methods of determining relative age by indicators such as the amount of tooth wear or changes in bone structure have yielded valuable information. Bird bands and other identifying marks also make age estimation possible. But one of the consequences of the fact that animals move is that very little is known about the life span of most species as they exist in nature.

The extreme claims of longevity that are occasionally made for one species or another have consistently been proven false when subjected to critical scrutiny. Although the maximum life span that has been observed for a particular species cannot be considered absolute, since a limited number of individuals at best has been studied, this datum probably provides a fair approximation of the greatest age attainable for this kind of animal under favourable conditions. Animals in captivity, which provide most of the records of extreme age, are exposed to far fewer hazards than those in the wild. In the accompanying table of maximum longevity, particular species have been so selected as to encompass the known range of longevity of other members of the taxonomic group to which they belong.

Maximum longevity of animals in captivity

Life span usually is measured in units of time. Although this may seem eminently logical, certain difficulties may arise. In cold-blooded animals in general, the rate of metabolism that determines the various life processes varies with the temperatures to which they are exposed. If aging depends on the expenditure of a fixed amount of vital energy, an idea first proposed in 1908, life span will vary tremendously depending on temperature or other external variables that influence life span. There is considerable evidence attesting at least to the partial cogency of this argument. So long as a certain range is not exceeded, cold-blooded invertebrates do live longer at low than at high temperatures. Rats in the laboratory live longest on a somewhat restricted diet that does not permit maximum metabolic rate. Of perhaps even greater significance is the fact that many animals undergo dormant periods. Many small mammals hibernate; a number of arthropods have life cycles that include periods during which development is arrested. Under both conditions the metabolic rate becomes very low. It is questionable whether such periods should be included in computing the life span of a particular organism. Comparisons between species, some of which have such inactive periods while others do not, are dangerous. It is possible that life span could be measured more adequately by total metabolism; however, the data that are necessary for this purpose are almost entirely lacking.

Length of life is controlled by a multitude of factors, which collectively may be termed environment, operating on a genetic system that determines how the individual will respond. It is impossible to list all the environmental factors that may lead to death. For analytical purposes it is, however, useful to make certain formal separations. Every animal is exposed to (1) a pattern of numerous events, each with a certain probability of killing the individual at any moment and, in the aggregate, causing a total probability of death or survival; (2) climatic and other changes in the habitat, modifying the frequency with which the various potentially fatal events occur; and (3) progressive systemic change, inasmuch as growth, reproduction, development, and senescence are characteristics intrinsic in the organism and capable of modifying the effects of various environmental factors.

Consider a group of similar animals of the same age. Although no two individuals can have precisely the same environment, let it be assumed that the environment of the group remains effectively constant. If the animals undergo no progressive physiological changes, the factors causing death will produce a death rate that will remain constant in time. Under these conditions, it will take the same amount of time for the population to become reduced to one-half its former number, no matter how many animals remain at the beginning of the period considered. The animals therefore survive according to the pattern of an accident curve. This is the sense in which many of the lower animals are immortal. Although they die, they do not age; how long they have already lived has no influence on their further life expectation.

Another group of animals may consist of individuals that differ markedly in their responses to the constant environment. They may be genetically different, or their previous development may have caused variations to arise. Those individuals that are most poorly suited to the new environment will die, leaving survivors that are better adapted. The same result can also be achieved in other ways. If the environment varies geographically, those individuals that happen to find areas in which existence can be maintained will survive, while the remainder will die. Or, as a result of their own properties, animals in a constant environment may acclimate in a variety of ways, thus adjusting to the existing conditions. The pattern of survival that results in each of these cases is one in which the death rate declines with time, as illustrated by the selectionacclimation curve.

In the absence of death from other causes, all members of a population may exist in their environment until the onset of senescence, which will cause a decline in the ability of individuals to survive. In a sense they can be considered to wear out as does a machine. Their survival is best described by individual differences among members of the population that determine the curvature of the survival line (wearing-out curve). The more the population varies, the less abrupt is the transition from total survival to total death.

Under the actual conditions of existence of animals the three types of survival (accident pattern, selectionacclimation pattern, wearing-out pattern) above all enter as components of the realized survival pattern. Thus in animals that are carefully maintained in the laboratory, survival is approximately that of the wearing-out pattern. Environmental accidents can be kept to a minimum under these conditions, and survival is almost complete during the major part of the life span. In all known cases, however, the early stages of the life span are characterized by a noticeable contribution of the selectionacclimation pattern. This must be interpreted as a result of developmental changes that accompany the early life of the individuals and of selective processes that operate on those organisms whose genetic constitutions are ill fitted for that environment.

In some of the larger mammals in nature, the existing evidence points to a similar survival pattern. In a variety of other animals, however, and including fishes and invertebrates, mortality in the young stages is so high that the selectionacclimation curve predominates. One estimate places the mortality of the Atlantic mackerel during its first 90 days of life as high as 99.9996 percent. Since some mackerel do live for several years, a mortality rate that decreases with age is indicated. Similar considerations probably apply to all those animals that have larval stages that serve as dispersal mechanisms.

When the postjuvenile portion of the life span is considered by itself, a number of animals for which such information has been gatheredincluding primarily fishes and birdshave survivorship curves that are dominated by the accident pattern. In these species in nature, death from old age apparently is rare. Their chance of surviving to an advanced age is so small that it may be statistically negligible. In modern times, human predation is a large factor in the mortality of these species in many cases. Since deaths from fishing and hunting are largely independent of age, once an animal has reached a certain minimum size, such a factor only makes the survival curve steeper but does not change its shape. One consequence of such increased mortality is that fewer old and large individuals are noticed in a population.

More complex survival patterns, such as the hypothetical one illustrated, undoubtedly exist. They should be looked for in those species in which extensive reorganization of the animal is part of the normal life cycle. In effect, these animals change their environment radically, in some cases several times during a lifetime. The frog offers a familiar example. During its period of early development and until shortly after hatching, the animal is subject to major internal, and some external, change. As a tadpole it is adjusted to an aquatic, herbivorous life. The metamorphosis to the terrestrial, carnivorous adult form is accompanied by varied physiological stresses that must be expected to produce a temporary increase in mortality rate. In some insects the eggs, larvae, pupae, and adults are exposed to and respond to quite different environments, and a survivorship pattern even more complex than that described by the composite curve may exist.

The same species will exhibit changed survival in different environments. In captivity an animal population may approach the wearing-out pattern; in its natural habitat survivorship may vary with age in a quite different way. Although one can assign a maximum potential life span to an individualwhile realizing that this maximum may not be attainedit is impossible to specify the survivorship pattern unless the environment is also specified. This is another way of saying that life span is the joint property of the animal and the environment in which it lives.

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New England Centenarian Study BUMC

Thursday, August 4th, 2016

New England Centenarian Study

To Discover the Secrets of A Long Healthy and Happy Life

Our two major studies are the New England Centenarian Study (founded 1995) and the multi-center Long Life Family Study (Boston Medical Center is one of 5 study sites),established in 2006.

We are actively seekingparticipants to be in the New England Centenarian Study. The criteria are simply subjects age 103+ years oldor 100+ years with siblings.

If you would like to contact the study, please call us at 888-333-6327 (toll free) where you will hear a menu tobe connected toa member of the research staff (please choose this option). You can alsoemail the study manager, Stacy Andersen PhD at stacy@bu.edu or the Principal Investigator, Thomas Perls MD, MPH at thperls@bu.edu.

The New England Centenarian Study is funded by:

The Martin A. Samowitz Foundation

Since 2006, the Long Life Family Studyhas beenconducting a prospective study of 5,000 subjects belonging to about 550 families that demonstrateparticularly unusualclustering for exceptional longevity. The Long Life Family Study is funded by and collaborates closely with The National Institute on Aging.

Pleasecall Dr. Thomas Perls toll-free at 888-333-6327 or email him at thperls@bu.edu if you or a family member wish to discuss our studies, find out more information regarding enrollment or to discuss supporting our studies.

MEDIA INQUIRES: Please contact

Jenny Eriksen Leary Manager of Media Relations Boston Medical Center 617-638-6841 jenny.eriksen@bmc.org

Genes play a critical and complex role in facilitating exceptional longevity. The genetic influence becomes greater and greater with older and older ages, especially beyond 103 years of age.

Citation: Genetic Signatures of Exceptional Longevity in Humans. Paola Sebastiani, Nadia Solovieff, Andrew T. DeWan, Kyle M. Walsh, Annibale Puca, Stephen W. Hartley, Efthymia Melista, Stacy Andersen, Daniel A. Dworkis, Jemma B. Wilk, Richard H. Myers, Martin H. Steinberg, Monty Montano, Clinton T. Baldwin, Josephine Hoh, Thomas T. Perls. PloS ONE 2012. DOI: 10.1371/journal.pone.0029848.

Many of the above genetic findings were replicated in a combination of 5 collaborating centenarian studies.

Citation: Meta-analysis of genetic variants associated with human exceptional longevity. Paola Sebastiani, Harold Bae1, Fangui X. Sun, Stacy L. Andersen, E. Warwick Daw, Alberto Malovini, Toshio Kojima, Nobuyoshi Hirose, Nicole Schupf, Annibale Puca, Thomas T Perls. Aging (Albany NY) 2013 September; 5(9): 653661. Published online 2013 August 24. PMCID: PMC3808698

In another paper published January, 2012, we have produced perhaps some of our most exciting findings to date. Early oninThe New England Centenarian Study, we thought that centenarians had to markedly delay or even escape age-related diseases like heart attacks, stroke, diabetes and Alzheimers, or else they would never be able to get to their very old ages. In fact, in 1980, a Stanford researcher named James Fries proposed the Compression of Morbidity hypothesis which states that as one approaches the limit of human life span, they must compress the time that they develop diseases towards the very end of their life and he proposed that people around the age of 100 do this. However, in 2003 we found that many of our centenarian subjects had age related diseases even before the age of 80 (about 43%, and whom we called survivors), after the age of 80 (about 42% and whom we called delayers) and lastly, those who had no mortality-associated diseases at age 100 (about 15% and whom we called escapers). The key though was that 90% of all of the centenarians were still independently functioning at the average age of 93 years. Somehow, despite the presence of diseases, people who become centenarians dont die from those diseases, but rather they are able to deal with them much better than other people and remain independently functioning more than 30 years beyond the age of 60. Therefore it seemed to us that for these study participants, it was not so much the compression of morbidity that was important to their survival, but rather a compression of disability.

In this current paper though, titled Health span approximates life span among many supercentenarians: Compression of morbidity at the approximate limit of life span, we have found that we just werent looking at old enough subjects when investigating Jim Fries hypothesis. As some of you know, over the past few years we have been working hard on recruiting and enrolling the most extreme old, supercentenarians who are people that live to 110 years and older. Once we enrolled our hundredth super-centenarian (by far the largest collection of supers in the world), we were able to investigate whether or not people who truly approach the limit of human lifespan actually compress their morbidity towards the end of their lives. Inour study of a reference group, nonagenarians (subjects in their nineties), centenarians (ages 100-104), semi-supercentenarians (ages 105-109) and supercentenarians(ages 110+), the subjects had progressively shorter periods of their lives spent with age-related diseases, from 17.9% of their lives in the referent group, to 9.4% in the nonagenarians and down to 5.2% in the supercentenarians. These findings support the compression of morbidity hypothesis and the idea that there truly is a limit to human life span 125 years. Also the supercentenarians were much more alike in terms of the markedly delayed age of onset of age-related diseases compared to the subjects age 100-104 who were quite heterogeneous. That homogeneity indicates they must have some factors (presumably genetic) in common that allow them to be so similar. We believe that our oldest subjects, ages 105+ years, give us the best chance and discovering these genes.

Citations: Health span approximates life span among many supercentenarians: Compression of morbidity at the approximate limit of life span Andersen SL, Sebastiani P, Dworkis DA, Feldman L, Perls T. J Gerontol A Biol Sci Med Sci 2012;67A:395-405.

Families Enriched for Exceptional Longevity also have Increased Health-Span: Findings from the Long Life Family Study. Paola Sebastiani, Fangui X. Sun, Stacy L. Andersen, Joseph H. Lee, Mary K. Wojczynski, Jason L. Sanders, Anatoli Yashin, Anne B. Newman, Thomas T. Perls. Front Public Health. 2013; 1: 38. Prepublished online 2013 August 16. Published online 2013 September 30. doi:10.3389/fpubh.2013.00038. PMCID: PMC3859985.

The New England Centenarian Study, along with collaborators at the Scripps Institute and the University of Florida, Gainesville, performed and published the first-ever whole genome sequence of a supercentenarian and actually not one super, but two, both over the age of 114 years and one was a man and the other a woman. As with our paper on the genetic signatures of exceptional longevity, we found here as well that centenarians have just as many genetic variants associated with diseases as the general population. However, they likely also have longevity-associated variants that counteract such disease genes, thus allowing for slower aging and increased resistance to age-related diseases.

In this paper we also found several genes that occurred in our published genetic prediction model which had coding regions that led to differences in gene function. These findings support the validity of the genetic prediction model. The New England Centenarian Study has posted the whole genome sequences of these two subjects on a data repository (called dbGaP) based at the National Institutes of Health. This will allow researchers from around the world to access all of the data and use them for their own research. Our hope is that these data will lead to important discoveries about genes that help delay or allow the escape from age related diseases like Alzheimers disease.

Citation: Whole genome sequences of male and female supercentenairnas, Both ages >114 years. Sebastiani P, Riva A, Montano M, Pham P, Torkamani A, Scherba E, Benson G, Milton JN, Baldwin CT, Andersen S, Schork NJ, Steinberg MH, Perls T. Frontiers in Genetics of Aging 2012;2.

There is a growing body of evidence for a substantial genetic influence upon survival to the most extreme ages. An important question is what would be the selection pressure(s) for the evolution of longevity associated genetic variants. The pressure to have a longer period of time during which women can bear children and therefore have more of them and therefore have greater success in passing ones genes down to subsequent generations could be one such pressure. This hypothesis is consistent with the disposable soma theory where the tradeoff in energy allocation between reproductive fitness and repair/maintenance functions can be delayed when longevity associated variants facilitate slower aging and the delay or prevention of age-related diseases that also adversely affect fertility. Several studies have noted an association between older maternal age and an increased odds of exceptional survival. The New England Centenarian Study assessed maternal age history in its sample of female centenarians and a birth-cohort-matched referent sample of women who survived to the cohorts average life expectancy. Women who gave birth to a child after the age of 40 (fertility assistance was not technologically available to this cohort) had a four times greater odds of being a centenarian. Numerous investigators are now searching for and investigating genes that influence reproductive fitness in terms of their ability to also influence rate of aging and susceptibility to age-related diseases.

Citations: Middle-aged mothers live longer.Perls TT, Alpert L, Fretts RC. Nature. 1997 Sep 11;389(6647):133.PMID: 9296486 [PubMed indexed for MEDLINE]

Extended maternal age at birth of last child and womens longevity in the Long Life Family Study.Sun F, Sebastiani P, Schupf N, Bae H, Andersen SL, McIntosh A, Abel H, Elo IT, Perls TT. Menopause. 2015 Jan;22(1):26-31. doi: 10.1097/GME.0000000000000276. PMID: 24977462. [PubMed in process]

The reappearance of procaine hydrochloride (Gerovital H3) for antiaging.Perls T. J Am Geriatr Soc. 2013 Jun;61(6):1024-5. doi: 10.1111/jgs.12278. No abstract available. PMID: 23772727. [PubMed indexed for MEDLINE]

Growth hormone and anabolic steroids: athletes are the tip of the iceberg.Perls TT. Drug Test Anal. 2009 Sep;1(9-10):419-25. doi: 10.1002/dta.87. PMID: 20355224 [PubMed indexed for MEDLINE]. Abstract: Professional Athletes misuse of anabolic steroids, growth hormone and other drugs are the tip of a very large, mostly ignored iceberg, made up of people who receive these drugs for such non-medical uses as body-building, school sports and anti-aging. Although these drugs are often used in combination, this article focuses on growth hormone. Fuelling the demand for these drugs are drug manufacturers, pharmacies, websites, clinics and their doctors.

New developments in the illegal provision of growth hormone for anti-aging and bodybuilding.Olshansky SJ, Perls TT. JAMA. 2008 Jun 18;299(23):2792-4. doi: 10.1001/jama.299.23.2792. No abstract available. PMID: 18560007 [PubMed indexed for MEDLINE]

DHEA and testosterone in the elderly.Perls TT. N Engl J Med. 2007 Feb 8;356(6):636; author reply 637. No abstract available. PMID: 17288051 [PubMed indexed for MEDLINE]

Hope drives antiaging hype.Perls TT. Cleve Clin J Med. 2006 Dec;73(12):1039-40, 1044. Review. No abstract available. PMID: 17190307 [PubMed indexed for MEDLINE]

Provision or distribution of growth hormone for antiaging: clinical and legal issues.Perls TT, Reisman NR, Olshansky SJ. JAMA. 2005 Oct 26;294(16):2086-90. No abstract available. PMID: 16249424 [PubMed indexed for MEDLINE]

Anti-aging quackery: human growth hormone and tricks of the trademore dangerous than ever.Perls TT. J Gerontol A Biol Sci Med Sci. 2004 Jul;59(7):682-91. PMID: 15304532 [PubMed indexed for MEDLINE]

The hype and the realitypart I.Olshansky SJ, Hayflick L, Perls TT. J Gerontol A Biol Sci Med Sci. 2004 Jun;59(6):B513-4. No abstract available. PMID: 15215255 [PubMed indexed for MEDLINE]

Antiaging medicine: what should we tell our patients? Perls T. Aging HealthApril 2010, Vol. 6, No. 2, Pages 149-154 , DOI 10.2217/ahe.10.11 (doi:10.2217/ahe.10.11)

To speak to someone about our research, please call our toll free number: 1-888-333-NECS (6327). Choose the option to speak with a member of our staff and you will be directed to the right person.

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The benefits of mucuna pruriens are … – Secrets of Longevity

Thursday, August 4th, 2016

The amazing herbal adaptogen that is mucuna pruriens is well known for it's wide range of tonifying, strengthening and all around beneficial properties. Perhaps it is best known as a potent herbal aphrodisiac. This Indian herb is known by many names, some of which are cowitch, cowhage, velvet beans, sea beans, buffalo beans, Kapikachu, Atmagupta and many more!

The prickly orange hairs on the outside of the pod cause severe itching when they come in contact with the skin. The doctrine of signatures can give us a clue here since the velvet bean's pods can be used to treat snake bite! The main medicinal effects attributed to mucuna pruriens come from the seeds, but the pod and its hairs and the plant's roots can also be used in herbal preparations. The velvet beans are "drift seeds", which is a name given to any seeds that can float away on ocean currents to grow in other regions of the world.

~Sweet, bitter and hot

~Heavy and unctuous (oily or greasing quality)

~Balancing to all three doshas! (a rare quality even amongst herbal adaptogens, ginseng and gotu kola are also known for this)

~Builds fertility in men (Increases semen volume, sperm count and sperm motility, better double up boys...)

~Very potent at increasing libido for both men and women

~Treats leucorrhoea (vaginal discharge), menorrhagia (excessive menstrual bleeding) and spermatorrhoea (involuntary ejaculation) which are all signs of leakage of Jing/Ojas, which is the foundational essence of health

~Helps men last longer sexually

~Used to build muscle in those who are weak

~Helps women increase lactation when breastfeeding

~Prevents and or lowers the effects of poisonous snake bites

~Treats Parkinson's disease

~Treats depression

~Treats insomnia and generally deepens sleep

~The hairs on the mucuna pod are used to treat several species of parasitic worms (excluding tapeworms)

~Helps digestion without increasing pitta (fire)

This link will take you to my favorite mucuna extract to use in smoothies and elixirs. Herbal extracts are easy to use and are extremely bio-available (absorbable) by the body.

If you prefer to work with whole herbs, you can get another amazing brand of Organic Mucuna Seed Powder by clicking this text link.

Mucuna pruriens contains 40mg/g of a compound called L-DOPA which is a direct precursor to the neurotransmitter dopamine. Dopamine works as one of the best natural hgh releasers (human growth hormone) by stimulating the pituitary gland to increase it's production. High levels throughout life of the body's own natural growth hormone (not hormones from the synthetic injections) is known as a major key for human longevity.

Dopamine also increases other youth hormones such as testosterone (it must be mentioned that these increases are never throwing the endocrine system out of balance, merely improving levels which tend to decrease over time in the average person). These resulting increased levels of these hormones from the use of mucuna pruriens extracts, powders and/or raw herbs explains its association with increasing libido, fertility, lean muscle mass, reducing signs of aging such as wrinkles and body fat, increasing bone density and even boosting overall energy levels.

Animal studies have shown powerful results such as a ten fold increase in mounting frequency by male rats! Also a decrease in post ejaculatory intervals have been observed, meaning that the animals being studied are more quickly able to return to "the task at hand". This could be due to the fact that mucuna pruriens extract basically cures premature ejaculation and the amount of downtown between ejaculations by reducing hypersensitivity in the genitals.

L-DOPA from mucuna pruriens has been shown to be safer and more effective for controlling Parkinson's disease than the pharmaceutical drugs Levodopa and Carbidopa. This is because the vast array of other chemical constituents that accompany L-DOPA in the unprocessed plant also play a role in its ability to manage Parkinson's disease. Since Parkinson's is in large part a deficiency of the neurotransmitter dopamine, this adaptogenic herb is crucial for anyone looking to manage this disease. It also looks to be very promising as a treatment for recovering drug addicts and people suffering from depression related illness, since these people have deficiencies of the neurotransmitters serotonin and dopamine (which the velvet beans contain and help balance in the human body).

The anti-snake venom effect of mucuna pruriens extract makes it a great tonic herb for outdoor enthusiasts to take daily. A variety of toxins from snake venom have been found to be reduced or prevented by the supplementation of this herb. Of course, one has to be taking it for it to work and it's best taken all the time since snake bites are completely unpredictable, yet obviously a risk for anyone poking around in the woods and other environments where these creatures hang out.

Enhanced lucid dreaming effects have been reported with the use of this amazing herb. Other chemicals in addition to L-DOPA found in mucuna pruriens that can have a mind altering effect include serotonin (5HT), oxitriptan (5-HTP), nicotine, N,N-DMT, bufotenin (5-HO-DMT) and 5-MeO-DMT. These would explain its noted effects as an entheogenic substance often found in Ayahuasca preparations (an Amazonian psychedelic shamanic herbal preparation). Do not be alarmed however, these chemicals are not in present in high enough quantities to cause "reality altering" effects, unless the herb happens to be combined with a sufficient amount of an MAOI 😉

This herbal adaptogen can also be used by anyone simply wanting to have fun. The abundance of bliss chemicals combined with the velvet beans adept ability at helping men last longer sexually and increasing libido for both genders makes this a safe and hilariously fun herb to use recreationally. It is also quickly becoming popular as a natural bodybuilding hgh secretagogue. This is much safer to use than synthetic human growth hormone which is quickly gaining a reputation as a carcinogenic "steroid-like" performance enhancer. The great thing with this herb is that it gets your body to make growth hormone itself and thus control the amount it makes and avoid the problems associated with elevated levels of hgh.

This link will take you to my favorite mucuna extract to use in smoothies and elixirs. Herbal extracts are easy to use and are extremely bio-available (absorbable) by the body.

If you prefer to work with whole herbs, you can get another amazing brand of Organic Mucuna Seed Powder by clicking this text link.

One of the most important secrets of longevity is maintaining optimal levels of key hormones as well as other phytochemical factors in your bloodstream. This article has already covered how mucuna pruriens is a natural growth hormone secretagogue (hgh) as well as a booster of other hormones of youth that tend to diminish with time. It has been found that blood plasma levels of some of the top antioxidants such as superoxide dismutase (SOD), catalase (an enzyme that prevents your hair from going gray!), glutathione (GSH) and ascorbic acid (vitamin C) are kept elevated in those who take this adaptogenic herbal aphrodisiac.

The powerful benefits of mucuna pruriens extract and whole herb should be temporarily avoided while pregnant or suffering from diabetes (it has blood sugar lowering properties). It should also be avoided by anyone with schizophrenia. If you choose to use adaptogenic herbs to manage or reverse any disease or disorder, always work with a knowledgeable health care practitioner. There is no specifically determined dosage for this herbal aphrodisiac, however most people agree on 15-30 grams a day for adults as a safe and effective amount.

Sources:

~http://ecam.oxfordjournals.org/cgi/content/abstract/nem171 ~http://herbal-powers.com/macunapruriens1.html ~http://www.herbalcureindia.com/herbs/mucuna-pruriens.htm ~http://en.wikipedia.org/wiki/Mucuna_pruriens ~http://www.ncbi.nlm.nih.gov/pubmed/18203263?itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum&ordinalpos=19 ~http://www.ncbi.nlm.nih.gov/pubmed/18973898?itool=EntrezSystem2.PEntrez.Pubmed.Pubmed_ResultsPanel.Pubmed_RVDocSum&ordinalpos=15

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At 100, The Secret of David Rockefellers Longevity …

Thursday, August 4th, 2016

Organ transplants.

Cannibalizing the dead.

Fitting for one of the worlds chiefSatanists, responsible forthe

deaths of millions.

Last week (June 12), David Rockefeller celebrated his 100th birthday without much ado.

He said the secret of his longevity was leading a simple life.

People often ask me this question and I always answer the same thing: Love life. Live a simple life, play with your kids, enjoy the things you have, spend time with good loyal friends.

He might have added that having a heart transplant every 6-7 years might have been a factor.

Rockefeller has had six heart transplants since 1976 when a car accident led him to suffer aheart attack. He has also had two kidney transplants in 1988 and 2004.

Just two months ago, the billionaire philanthropist had his sixth heart transplant.

The heart transplant surgery, which lasted 6 hours and was done by a team of private surgeons at his principal residence, on the family estate in Pocantico Hills, New York, doesnt seem to have tired the legendary business man known for his dynamism and cunningness.

With all the carnage due to the Illuminati agenda, there are plenty of beating hearts for him to pillage.

Note:

Memoirs (2003)[edit]

ISBN-13: 978-0812969733

For more than a century, ideological extremists at either end of the political spectrum have seized upon well-publicized incidents such as my encounter with Castro to attack the Rockefeller family for the inordinate influence they claim we wield over American political and economic institutions. Some even believe we are part of a secret cabal working against the best interests of the United States, characterizing my family and me as internationalists and of conspiring with others around the world to build a more integrated global political and economic structure one world, if you will. If that is the charge, I stand guilty, and I am proud of it.

The anti-Rockefeller focus of these otherwise incompatible political positions owes much to Populism. Populists believe in conspiracies and one of the most enduring is that a secret group of international bankers and capitalists, and their minions, control the worlds economy. Because of my name and prominence as head of the Chase for many years, I have earned the distinction of conspirator in chief from some of these people.

Populists and isolationists ignore the tangible benefits that have resulted in our active international role during the past half-century. Not only was the very real threat posed by Soviet Communism overcome, but there have been fundamental improvements in societies around the world, particularly in the United States, as a result of global trade, improved communications, and the heightened interaction of people from different cultures. Populists rarely mention these positive consequences, nor can they cogently explain how they would have sustained American economic growth and expansion of our political power without them.

Ch. 27 : Proud Internationalist, p. 406

https://en.wikiquote.org/wiki/David_Rockefeller

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At 100, The Secret of David Rockefellers Longevity ...

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How Long Will I Live? – Life Expectancy Calculator

Friday, October 23rd, 2015

We have been working to update the interface of the tool and integrate the latest available data into our calculations. Shortly, this version of the calculator will be replaced. The beta version of the updated calculator is available here. Feedback? Fill out this quick survey to let us know. Fill in the following form then click the button labeled "Calculate Life Expectancy". For values which you are unsure of, leave it blank or choose option 'don't know'; For zero values, enter "0", DO NOT leave them blank If you're in a hurry, try our Short life expectancy calculator.

I am year old male female white nonwhite My height is inches (NOTE!!! Only input inches: Eg. 5'8" = 68 inches) My weight is pounds I expect to have less than 10 10 to 11 more than 11 don't know years of education My family's total income for the past 12 months is dollars I expect that for most of my life I will be married not married don't know Compared to other people of the same age and sex as me, I am in the 1st (least fit) 2nd 3rd 4th 5th (fittest) don't know quintile of fitness(refer to Fitness Table) I do not do don't know have at least one first degree relative (parents, sibling, children) who has a history of heart diseases I do not do don't know have at least one first degree relative (parents, sibling, children) who has a history of prostate cancer I do not do don't know have at least one first degree relative (parents, sibling, children) who has a history of breast cancer I do not do don't know have at least one first degree relative (parents, sibling, children) who has a history of colorectal cancer I do not do don't know have at least one first degree relative (parents, sibling, children) who has a history of stomach cancer I do not do don't know have at least one first degree relative (parents, sibling, children) who has a history of lung cancer None One Two or more don't know of my first degree relative (parents, sibling, children) has a history of diabetes I do not do don't know have at least one first degree relative (parents, sibling, children) who has a history of stroke I reside in Alabama Alaska Arizona Arkansas California Colorado Connecticut Delaware District of Columbia Florida Georgia Hawaii Idaho Illnois Indiana Iowa Kansas Kentucky Louisiana Maine Maryland Massachusetts Michigan Minnesota Mississippi Missouri Montana Nebraska Nevada New Hampshire New Jersey New Mexico New York North Carolina North Dakota Ohio Oklahoma Oregon Pennsylvania Rhode Island South Carolina South Dakota Tennessee Texas Utah Vermont Virginia Washington West Virginia Wisconsin Wyoming don't know I have not have don't know been diagnosed with asthma I have not have don't know been diagnosed with diabetes My diastolic blood pressure (the smaller/bottom number- an average adult's is about 80) is mmHg

I smoke cigarettes per day My spouse smokes cigarettes per day I have 0 or negligible less than 1 1 2 to 3 4 or more don't know drinks per day I travel thousand miles per year in an automobile The driver of the automobile which I most frequently travel in is a male female don't know The age of the driver of the automobile which I most frequently travel in is years I do not do don't know regularly wear seat belts when travelling in a automobile The automobile which I most frequently travel in does not does don't know regularly keep to speeds appropriate to road conditions The driver of the automobile which I most frequently travel in is sometimes never don't know drunk while driving Of the 10 things listed in the Stress List, of them happened to me in the past 12 months I am a sedentary person occasional exerciser conditioning exerciser don't know I work in the mining construction transportation/public utilities agriculture/forestry/fishing public administration manufacturing retail trade services wholesale trade finance/real estate all others don't know industry My father worked in a non-manual manual don't know job My first regular occupation is a non-manual manual don't know job My current occupation is a non-manual manual don't know job Of the 5 types of food in the Dietary Diversity List, on average I consume types more less don't know than 10% of my energy intake comes from fat I am not am don't know among the 15% most depressed of the population I have had sexual partners in the past 12 months For most of my sexual encounters, I do not do don't know use condoms On average, I have hours of sleep a day

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The Big Physical: Where to Go, What to Get | Departures

Friday, October 9th, 2015

While getting old may beat the alternative, living in denial is a comfortable choiceand a lot more pleasant than your first colonoscopy. The annual checkup at your doctors office covers the basics, but a truly thorough medical exam will involve trips to several offices, with the usual long waits and patients stacked up like planes over Chicagos OHare. Two years ago one of my closest friends died from a coronary that absolutely could have been prevented had he only taken the time from his crazy work schedule and family obligations for a checkup.

With baby boomers starting to age, there has been a proliferation of facilities offering comprehensive "executive" physicals. Having just turned 50 myself, I figured this might be a good time to see how the examsand my bodyhave evolved. Among the best-known options, the Mayo and Cleveland clinics, along with the Johns Hopkins, Duke, UCLA, and Stanford units, are all associated with major hospitalswhich is useful should serious complications be found or immediate treatment be recommended. According to the Mayo Clinic, less than 5 percent of its exams revealed potentially life-threatening conditions and almost half exposed risk factors for serious illness. These centers draw patients from all over the world and focus not just on medical needs but a lifestyle approach as well. The Cleveland Clinic even offers programs through the Canyon Ranch spa resorts. Also available are a number of private diagnostic centers that only perform physical exams, such as the Princeton Longevity Center in New Jersey and Texass Cooper Clinics. I chose the PLC strictly for its convenience, but some might be more comfortable going to a hospital. Others prefer to go the true luxury route and turn their physical into a spa holiday or fit it into a resort vacation. To me, though, the chance of getting some bad medical news before my kelp facial could be a real buzz kill.

As at most centers, the day in the PLC started at the scanning facility, replete with state-of-the-art, 64-slice CT machines. I was then shuttled around the main office, given every sort of workup imaginable, and promised a full report on my mortality within hours. Some centers recommend full-body CT scans for patients over 45. While they can be lifesavers, they are also controversial. Dr. Reginald Munden, interim chair of the department of Diagnostic Radiology at the University of Texas M. D. Anderson Cancer Center, says, "Im not comfortable with a shotgun approach to screening," citing the limited sensitivity of scans to common cancer lesions and the small but significant increase in cancer risk from more radiation exposure. There is also the issue of false positives: Up to 80 percent of such tests may find something that can lead to what Munden calls a spiral of expensive and anxiety-provoking follow-up scans with few meaningful results. Interestingly, just as risky can be false negativesbeing told the scan shows no problems to be concerned about. This can lead some people to continue damaging behavior, such as smoking, or to ignore new symptoms.

Of course, there are many stories of serious problems being caught. Doctors at the clinics have received numerous e-mails from patients whose renal cancer or developing aneurysm was detected early, possibly saving their lives. In fact, the day I was at the PLC an exinvestment banker discovered, after 53 years, that he was born with only one kidney. Balancing the risks against the benefits of a scan is a personal decision, and any clinic should provide you with plenty of information to allow you to make an educated evaluation. Dr. Richard S. Lang, who heads the section of preventive medicine at the Cleveland Clinic, explained that while "the scans may not be for everyone," they certainly "offer additional information to a patients profile" and, so long as the findings are managed properly, can be of great valueeven if just as a baseline for future exams.

After the CTs and blood tests are completed, you can expect a very detailed and unrushed physical from a senior staffer instead of the usual Cliffs Notes version offered by your GP. Every aspect of your medical history will be checkedyou will be asked to send copies of all available reports in advanceand any issues, aches, or pains considered. I then had a challenging cardiovascular stress test and gym session with senior exercise physiologist Chris Volgraf. A highly educated trainer and professional strength coach, he completely changed my perspective on my own workout regimen and showed me a series of warm-ups and exercises designed to make my gym time safer, more efficient, and more effective.

Lunch was followed by a vision and hearing test, a bone density and body composition scan, and a meeting with a nutritionist, who evaluated the three-day food log I had been asked to keep.

When all the tests were done, the biggest difference between a full day at one of the clinics and a quick routine maintenance check became clear: A senior doctor sits down with you and reviews the results of every test in detail and their implications. Seeing rotating, 3-D, full-color computer images of your internal organs can be disquieting. Dr. David Fein, medical director and founder of the PLC, told me that one of the most effective tools in convincing patients to modify their unhealthy lifestyle is showing them their blocked arteries or the vascular fat wrapped around their vital organs in high-definition splendor. "You can point to it and say Thats a picture of your heart attack, or your type 2 diabetes diagnosis in five years. " Pushing the predictive envelope, the Duke Executive Health Program will soon offer a cutting-edge genomic DNAbased assessment that screens for certain genetic risks.

I left the PLC with a hefty binder, complete with test results and a disc containing copies of my scans, to give to my personal physician. In my opinion anyone who can afford it, or whose employer or insurer will pay, should immediately sign up for a visit to one of the top clinics and return at reasonable intervals. The Mayo Clinic quotes studies showing that companies can save as much as 20 percent in additional medical claims and 45 percent in extra sick days taken by executives who have regularly undergone thorough physicals. Its unlikely you have ever gotten so complete and detailed a review of not just the results but also the meaning of routine medical tests, as well as specific and practical advice on diet and exercise. While affiliation with a hospital or a major medical clinic is obviously a plus, the most important thing a diagnostic center can do for you is get you in the door. So if traveling makes you less likely to go, pick a center nearby.

centerforpartnershipmedicine.com Location: Chicago, IL Program:1 day Approx. Cost: $3,000$6,000

clevelandclinic.org; executivehealthprogram.com Location: Cleveland, OH; Weston, FL; Toronto; Canyon Ranch (AZ, MA) Program:13 days Approx. Cost: from $3,000

cooperaerobics.com/clinic Location: Dallas and McKinney, TX Program:1 day Approx. Cost: $1,800$4,000

dukeexechealth.org Location: Durham, NC Program:1 day Approx. Cost: $2,800

execmd.com Location: Menlo Park, CA Program:1/2 day Approx. Cost: $2,000$2,600

hopkinsmedicine.org/gim/clinical/executive_health Location: Baltimore, MD Program:1 day Approx. Cost: $1,800$2,200

mayoclinic.org/executive-health Location: Rochester, MN; Scottsdale, AZ; Jacksonville, FL Program:12 days Approx. Cost: $1,500$6,000

theplc.net Location: Princeton, NJ Program:1 day Approx. Cost: $3,250

exechealth.ucla.edu Location: Los Angeles, CA Program:1 day Approx. Cost: from $2,400

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Comprehensive Health Exam :: Princeton Longevity Center

Wednesday, October 7th, 2015

Announcing Our Newest Location in Fairfax, Virginia Call Us for Details!

The Princeton Longevity Comprehensive Exam takes your health beyond the "Annual Physical".

With the latest technology, combined with caring experts who take the time to fully evaluate your health, the Princeton Longevity Center Comprehensive Exam can detect early cancers, heart disease, aneurysms and the "silent killers" that are often missed in a typical physical exam or routine blood tests.

Youll get the latest diagnostic, screening and imaging technologies to assess the state of your health and the detailed information you need to optimize the quality of your future years.

The scope of your examination and the amount of useful information you come away with is dramatically more revealing than a standard physical exam or consultation. The effect on your future can be dramatic.

Our proactive approach to illness and premature aging will help you identify your risk of future disease and create a personalized program to improve your health and maintain an active lifestyle. You'll learn how simple, easy changes in nutrition, fitness, lifestyle and natural supplements can effectively prevent most Cancers and Heart Disease.

You'll receive this total, in-depth evaluation in one day and one place. We'll make you comfortable and look after you every step of the way.

Best of all, you'll get the peace of mind that comes from knowing that you are doing all you can to take care of your health- for yourself and for the people who care about you.

Request an Appointment or More Information

Learn More About the Exam with Frequently Asked Questions About The Comprehensive Exam

Comprehensive Exam Components

Comprehensive Exam Options and Electives

Find out how to use your Health Savings Account or Flex Account for A Comprehensive Exam

The Road Map

We'll explain the results of your evaluations, provide you with a clear understanding of your health issues and create a detailed, customized treatment plan for you. You'll walk out with a practical "road map" you can follow to maintain and enhance your health and fitness on a daily basis.

At the Princeton Longevity Center, we are your partners in long-term health care. When you walk out of our office, our involvement in your well-being does not end, and we will continue to monitor your progress through a choice of Follow-Up Options. This is not just an exam, it's the start of your plan for staying well for the years to come.

The Rewards

A Comprehensive Exam lets you take control of your health. If you are worried about your risk of heart disease, cancer or other health problems, a Princeton Longevity Center Comprehensive Exam can give you the Peace of Mind that comes from knowing that all is well.

Youll know whether or not you are really at risk for heart attack, cancer, diabetes, premature aging or other significant health issues. And, you will find out what you can do about those risks. You'll learn how to prevent cancer, heart disease, diabetes and many other diseases from affecting your future.

You'll have the opportunity to anticipate and prevent illness and premature aging. You'll learn the simple, easy steps that you can take to prevent cancer, heart attacks, diabetes, lung disease, and more. We will show you how you can improve your nutrition and fitness so that your future years are healthier and more active.

The Princeton Longevity Center is focused on helping you with healthy living at the forefront of new medical knowledge and diagnostic technologies. So, you can stop worrying and know that you are doing all you can to optimize your health for today and the years to come.

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Princeton Longevity Center

Wednesday, October 7th, 2015

Sensation 64-Slice CT High Definition Coronary Artery Scans

I don't want to know if something is wrong. Won't the scan make me too nervous?

What type of scaner is used?

What happens during the scan?

What about the radiation exposure?

How long does a scan take?

Do I need to fast?

Do I need a referral from my physician?

Is the scan covered by my insurance?

How accurate is a Heart Scan?

My cholesterol level is normal. Should I still have a Heart Scan?

What if I already know I have heart disease?

I was told the scan can't detect "soft" plaque. Does that matter?

I already passed a stress test. Should I still have a scan?

How does the scan detect coronary artery disease?

What about blood tests like C-Reactive Protein and Homocysteine?

Is this the same scan that I saw on Oprah or in the newspapers?

What do I do with my test results?

What makes the Princeton Longevity Center's scans different from all the other scanning centers that I have seen or heard advertised?

Request an Appointment or More Information Back to Coronary Artery Scan Page

I don't want to know if something is wrong. Won't the scan make me too nervous?

Heart disease is extremely treatable, especially if found early. If your scan shows that you are at risk for developing coronary artery disease, we can show you how simple changes or treatments can dramatically alter your risk and lessen the chances that you will have a heart attack. Avoiding a scan will not stop the disease. Knowing what simple steps you can take will make a big difference in the quality of the rest of your life.

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What type of scanner is used?

Our scans are done with a Siemens Definition AS 64 Slice CT Scanner. This state of the art scanner uses very low radiation doses and produces exceptional detail, as small as 1/50th of an inch. The scans are also extremely fast, usually less than 10 seconds, so there is no need for prolonged breath-holds. The scanner is completely non-claustrophic.

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What happens during the scan? Is it uncomfortable?

64 Slice CT scans are quick, painless and safe. You lie on a table and hold your breath for a few seconds. There are no needles or dyes; it is not claustrophobic and you usually don't even have to remove your clothes.

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What about the radiation exposure?

There has been a lot of attention in the media surrounding the issue of radiation exposure. We keep our doses as low as possible. Our Coronary Calcium Heart Scans generally involve a dose of about 1 mSv, an amount much lower than is typically cited in the media. According to the US Nuclear Regulatory Commission the risk of 1 mSv is about 1 in 25,000 or, put another way, it raises your cancer risk from 20.000% to just 20.004%. On the other hand, your risk of dying from cardiovascular disease that goes undetected and untreated is 36%. We believe that the potential benefit of early detection of cardiovascular or other diseases more than offsets the extremely small potential risk of this level of radiation exposure.

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How long does a scan take?

The heart, lung or whole body scans take just a few seconds. We will spend some time explaining the procedure to you before the scan. You will get your results within a few mintues. Altogether, you should plan on being at our center about 30 minutes.

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Do I need to fast?

No. There is no special preparation or fasting required for heart, lung or whole body scans.

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Do I need a referral from my physician?

No. While many physicians do refer their patients for these scans, we do not require physician referrals. However, we do encourage you to discuss the test with your physician. With your consent, we will send a copy of your results to your physician along with any recommendations for further treatment or testing.

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Is the scan covered by my insurance?

Most private insurance plans will notcover coronary artery scans . In some cases, the coverage will depend on whether the test is done solely as a preventive screening test or to help your doctor make a decision about your treatment.

Medicare Beneficiaries please note: In general, Medicare does not cover preventive services. Coronary Arteries Scans (CPT Code 75571) are specifically a non-covered service. You will not be able to receive any reimbursement from Medicare for these services.

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How accurate is the Coronary Artery Scan?

It is VERY accurate. There are no false-positives. The presence of calcium in the coronary artery means there is plaque there. The relationship between calcium in the artery and the risk of a heart attack has been well established in numerous medical studies. The more calcium present the higher your risk of a heart attack. The absence of any calcium on your scan means you are not at risk for a heart attack with a 99.9% certainty.

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My cholesterol level is normal so why should I bother with a coronary scan?

Study after study has shown that cholesterol levels do NOT accurately predict who will have a heart attack. In fact, 70% of heart attacks occur in people with "normal" cholesterol levels. Even if your cholesterol level is below the current guidelines you can still develop coronary artery disease. With coronary artery scans we can tell if your current cholesterol level is safe for you or if it needs to be lower.

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What if I already know I have heart disease?

Coronary artery scans are useful not only in early detection of disease but also for following the course of the disease. The scan can be used to monitor your treatment with diet, medication, etc. If your treatment is working, the scan will show the improvement. If it is not working, serial scans can show the continuing progression of the coronary artery disease and alert your physician to the need for more aggressive or alternative treatments.

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I was told coronary artery scans can't detect "soft plaque" and isn't that really where the danger is?

While it is true that a coronary artery scan only shows "calcified plaque" and not "soft plaque" (plaque that has not yet calcified), that does not make the scan any less useful. Calcified plaque serves as a marker for the presence of soft plaque. Numerous studies have proven that the coronary artery scan is the most accurate and sensitive predictor of your risk of having a heart attack or significant coronary artery disease. If you don't have any calcified plaque the likelihood that there is any soft plaque is extremely small. No other non-invasive test can detect the presence of early coronary artery disease as well as coronary scans.

If you have a high level of calcified plaque or your clinical history warrants further investigation, a CT Angiography will very reliably detect the presence of soft plaque and measure whether there is a signficant blockage in the arteries.

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I already passed a stress test. Should I still have a scan?

Stress tests only show an abnormality if you have an artery that is at least 65-80% blocked. At that point, treatment may require surgery or angioplasty. And, 70% of heart attacks happen in arteries that are less than 50% blocked and a stress test would not have shown anything wrong. The coronary artery scan can detect developing blockages much earlier, often many years before anything is detectable on a stress test. At that point, treatment with minor changes in diet and lifestyle along with nutritional supplements or medications can be very effective in preventing any worsening of the disease. That can allow you to avoid surgery or invasive procedures later on.

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How does the scan detect coronary artery disease?

The coronary artery scan detects the presence of calcium deposits in the wall of the arteries. These are often present long before blockages have become severe enough to be found by other means such as stress tests. Coronary artery calcium deposits are a "clinical marker" for coronary artery disease. The more calcium present the higher the risk that you may have a heart attack.

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What about blood tests like C-Reactive Protein or Homocysteine levels? Can't they tell if I have heart disease?

There is currently NO blood test that is able to tell if you have coronary artery disease. Blood tests can only estimate your probability of getting coronary disease. Many people with high levels of cholesterol, C-Reactive Protein or Homocysteine do not have coronary artery disease. At the same time, it is common to still get coronary disease even if those tests are normal. Relying solely on those tests means treating a lot of people who do not actually need to be treated and missing a lot of cases where heart attacks could have been prevented. Coronary scans look directly at your own arteries to see if you have a problem or not. Then, treatment is based on what is actually going on in your arteries instead of a statistical "guess".

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Is this the same scan that I saw on Oprah or in the newspapers?

Yes. 64-Slice CT Scans have been prominently featured on many national talk shows and in numerous magazines and other publications.

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What do I do with my test results?

You will get a copy of your test results along with a detailed explanation of the significance of the restuls immediately after your scan. We strongly encourage you to share these results with your physician to seek his/her advice as to any additional treatment that may be recommended.

Our Full Body Scans include a consultation with one of our expert physicians who will also explain the significance of the heart scan results. If you have a Full Body Scan (either with your heart scan or as part of a Comprehensive Exam) you will also get a preliminary review, pending the final report by our radiologist. If you are referred by your physician, the report will be sent to your physician and further management should be discussed with your doctor. If you are not referred by a physician, the discussion of your scan results will include general guidelines for steps you can take to help lower your future health risks.

Imaging studies and other medical tests may reveal findings that require additional evaluation or treatment. THe Princeton Longevity Center does not offer Primary Care or on-going medical management. All findings related to any tests performed at Princeton Longevity Center should be discussed by you with your Primary Care or Family Physician. If additional testing or treatment is warranted this can be arranged through your personal physician. A copy of your results will be sent to any physician you designate.

Back to TOP Request an Appointment or More Information Back to Coronary Artery Scan Page

What makes the Princeton Longevity Center's scans different from all the other scanning centers that I have seen or heard advertised?

For cardiac scans, the 64-Slice CT Scanner is the "gold standard." The Princeton Longevity Center is the region's leader in Cardiac Imaging and prevention. We don't just do Cardiac CT, we TEACH it. Our physicians have trained over a thousand physicians from around the world at our renowned Cardiac CT Training Course. Our scans are read twice, including our premier experts in cardiac imaging and radiologists expert in body scans. No other cardiac imaging center in the area offers this level of expertise in the interpretation of your results.

The Princeton Longevity Center is the only scanning center in the region that goes beyond scanning. Our staff of expert physicians, nutritionists and exercise physiologists are available to show you how to take the next step with our Comprehensive Exam program.

After we explain your results to you in-depth, our comprehensive preventive medicine center can offer you the tools and resources you need to immediately start to take a proactive role in your health. We work with you and your family physician to design a plan that will quickly start to lower your risk of heart attack and many other life-threatening diseases.

Other scanning centers may be able to tell you if there is a problem. The Princeton Longevity Center will tell you what you can do about the problem and help you take the next step to a healthier future.

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Product Longevity|Freescale

Wednesday, October 7th, 2015

Microcontrollers Kinetis K K02 - April 2014 Microcontrollers Kinetis K K10 - July 2011 Microcontrollers Kinetis K K11 - August 2012 Microcontrollers Kinetis K K12 - August 2012 Microcontrollers Kinetis K K20 - July 2011 Microcontrollers Kinetis K K21 - August 2013 Microcontrollers Kinetis K K22 - April 2014 Microcontrollers Kinetis K K24 - January 2014 Microcontrollers Kinetis K K26 - May 2015 Microcontrollers Kinetis K K30 - July 2011 Microcontrollers Kinetis K K40 - July 2011 Microcontrollers Kinetis K - K50 July 2011 Microcontrollers Kinetis K - K51 July 2011 Microcontrollers Kinetis K K60 - July 2011 Microcontrollers Kinetis K K63 - January 2014 Microcontrollers Kinetis K K64 - January 2014 Microcontrollers Kinetis K K65 - May 2015 Microcontrollers Kinetis K K66 - May 2015 Microcontrollers Kinetis K K70 - July 2011 Microcontrollers Kinetis L KL02 - June 2013 Microcontrollers Kinetis L KL03 - July 2014 Microcontrollers Kinetis L KL04 September 2012 Microcontrollers Kinetis L KL05 September 2012 Microcontrollers Kinetis L KL13 - May 2015 Microcontrollers Kinetis L KL14 - June 2012 Microcontrollers Kinetis L KL15 - June 2012 Microcontrollers Kinetis L KL16 - October 2013 Microcontrollers Kinetis L KL17 - September 2014 Microcontrollers Kinetis L KL24 - June 2012 Microcontrollers Kinetis L KL25 - June 2012 Microcontrollers Kinetis L KL26 - October 2013 Microcontrollers Kinetis L KL27 - September 2014 Microcontrollers Kinetis L KL33 - September 2014 Microcontrollers Kinetis L KL34 - July 2013 Microcontrollers Kinetis L KL36 - July 2013 Microcontrollers Kinetis L KL46 - July 2013 Microcontrollers Kinetis E KE02 - August 2013 Microcontrollers Kinetis E KE04 - March 2014 Microcontrollers Kinetis E KE06 - March 2014 Microcontrollers Kinetis V KV10 - April 2014 Microcontrollers Kinetis V KV31 - April 2014 Microcontrollers Kinetis V KV40 - April 2014 Microcontrollers Kinetis V KV45 - April 2014 Microcontrollers Kinetis V KV46 - April 2014 Microcontrollers Kinetis M KM14 - November 2013 Microcontrollers Kinetis M KM33 - November 2013 Microcontrollers Kinetis M KM34 - November 2013 Microcontrollers Kinetis W KW01 - March 2014 Microcontrollers Kinetis W KW21 - December 2013 Microcontrollers Kinetis W KW22 - December 2013 Microcontrollers Kinetis W KW24 - December 2013 Microcontrollers 8-bit RS08 RS08KA2/8 - April 2006 Microcontrollers 8-bit RS08 RS08LA8 - October 2008 Microcontrollers 8-bit RS08 RS08LE4 - January 2009 Microcontrollers 8-bit S08 S08QA4 - December 2005 Microcontrollers 8-bit S08 S08QG8 - December 2005 Microcontrollers 8-bit S08 S08QD4 - July 2006 Microcontrollers 8-bit S08 S08JM60/32 - October 2007 Microcontrollers 8-bit S08 - S08QE128/32/8 October 2007 Microcontrollers 8-bit S08 MC9S08SF4 - January 2008 Microcontrollers 8-bit S08 S08SH32/8 - March 2008 Microcontrollers 8-bit S08 S08AC128/16/60 - May 2008 Microcontrollers 8-bit S08 S08QB8 - October 2008 Microcontrollers 8-bit S08 - S08LL16/64 October 2008 Microcontrollers 8-bit S08 S08JS16 - November 2008 Microcontrollers 8-bit S08 S08SE8 - November 2008 Microcontrollers 8-bit S08 S08FL16 - February 2009 Microcontrollers 8-bit S08 S08SV16 - April 2009 Microcontrollers 8-bit S08 - MC9S08LH June 2010 Microcontrollers 8-bit S08 - MC9S08JE September 2010 Microcontrollers 8-bit S08 - MC9S08MM September 2010 Microcontrollers 8-bit S08 S08GW - January 2011 Microcontrollers 8-bit S08 S08P - November 2011 Microcontrollers 16-bit DSC MC56F8013/14 - April 2005 Microcontrollers 16-bit DSC MC56F802X - August 2007 Microcontrollers 16-bit DSC MC56F8006/2 - March 2009 Microcontrollers 16-bit DSC MC56F803X - July 2009 Microcontrollers 32-bit DSC MC56F84xx - June 2012 Microcontrollers 32-bit DSC MC56F827xx - November 2013 Microcontrollers 32-bit ColdFire MCF521X - September 2005 Microcontrollers 32-bit ColdFire MCF523X - September 2005 Microcontrollers 32-bit ColdFire MCF5207/8 - November 2006 Microcontrollers 32-bit ColdFire - MCF537X January 2007 Microcontrollers 32-bit ColdFire - MCF532X January 2007 Microcontrollers 32-bit ColdFire MCF5253 - February 2007 Microcontrollers 32-bit ColdFire MCF5223x - March 2007 Microcontrollers 32-bit ColdFire MCF51QE128/32 - June 2007 Microcontrollers 32-bit ColdFire MCF521XX - June 2007 Microcontrollers 32-bit ColdFire MCF5445x - September 2007 Microcontrollers 32-bit ColdFire MCF5227x - March 2008 Microcontrollers 32-bit ColdFire MCF51JM - July 2008 Microcontrollers 32-bit ColdFire MCF51AC256 - November 2008 Microcontrollers 32-bit ColdFire MCF5225x - November 2008 Microcontrollers 32-bit ColdFire MCF51CN - March 2009 Microcontrollers 32-bit ColdFire MCF51EM256 - May 2010 Microcontrollers 32-bit ColdFire - MCF5441x September 2010 Microcontrollers 32-bit ColdFire - MCF51JE September 2010 Microcontrollers 32-bit ColdFire - MCF51MM September 2010 Microcontrollers MPC5xxx 32-bit Power Architecture - MPC5125YVN400 April 2010 Processors i.MX Family i.MX233 - August 2009 Processors i.MX Family i.MX25 i.MX25 (Auto) June 2009 Processors i.MX Family i.MX27 - June 2007 Processors i.MX Family i.MX28 i.MX28 (Auto) September 2010 Processors i.MX Family i.MX31 - June 2005 Processors i.MX Family i.MX35 i.MX35 (Auto) October 2008 Processors i.MX Family i.MX50 - July 2011 Processors i.MX Family i.MX51 - November 2009 Processors i.MX Family i.MX53 i.MX53 (Auto) February 2011 Processors i.MX 6 Series i.MX 6UltraLite - September 2015 Processors i.MX 6 Series i.MX 6SoloLite - November 2012 Processors i.MX 6 Series i.MX 6SoloX i.MX 6SoloX (Auto) February 2015 Processors i.MX 6 Series i.MX 6Solo i.MX 6Solo (Auto) November 2012 Processors i.MX 6 Series i.MX 6Dual i.MX 6Dual (Auto) November 2012 Processors i.MX 6 Series i.MX 6DualLite i.MX 6DualLite (Auto) November 2012 Processors i.MX 6 Series i.MX 6Quad i.MX 6Quad (Auto) November 2012 Processors Vybrid Vybrid F Series - June 2013 Processors Vybrid - Vybrid R Series December 2014 Processors QorIQ - LS1020A/22A September 2013 Processors QorIQ - LS1021A September 2013 Processors QorIQ P1010/14 - November 2010 Processors QorIQ P1020/11 - June 2008 Processors QorIQ P1021/12 - December 2009 Processors QorIQ P1022/13 - August 2011 Processors QorIQ P1023/17 - August 2010 Processors QorIQ P1024/15 - December 2009 Processors QorIQ P1025/16 - December 2009 Processors QorIQ P2020/10 - June 2008 Processors QorIQ P2040/41 - May 2011 Processors QorIQ P3041 - June 2010 Processors QorIQ P4040 - September 2009 Processors QorIQ P4080 - June 2008 Processors QorIQ P5020/10 - June 2010 Processors QorIQ P5040/21 - May 2012 Processors QorIQ - T1024 April 2014 Processors QorIQ - T1040/42 October 2012 Processors QorIQ - T2080 June 2012 Processors QorIQ - T2081 October 2012 Processors QorIQ T4160 - February 2012 Processors QorIQ T4240 - February 2012 Processors QorIQ Qonverge BSC9131 - August 2011 Processors QorIQ Qonverge BSC9132 - August 2011 Processors QorIQ Qonverge B4860 - February 2012 Processors Crypto Coprocessors C29x - May 2013 Processors Host Processors MPC7448 - February 2004 Processors Host Processors MPC8641D - September 2004 Processors Host Processors MPC8640D - July 2008 Processors PowerQUICC II Pro MPC8358 - March 2005 Processors PowerQUICC II Pro MPC8360 - March 2005 Processors PowerQUICC II Pro MPC8347 - June 2005 Processors PowerQUICC II Pro MPC8349 - June 2005 Processors PowerQUICC II Pro MPC8321 - May 2006 Processors PowerQUICC II Pro MPC8323 - May 2006 Processors PowerQUICC II Pro MPC8313 - November 2006 Processors PowerQUICC II Pro MPC8314 - September 2007 Processors PowerQUICC II Pro MPC8315 - September 2007 Processors PowerQUICC II Pro MPC8378 - September 2007 Processors PowerQUICC II Pro MPC8379 - September 2007 Processors PowerQUICC II Pro MPC8377 - September 2007 Processors PowerQUICC II Pro MPC8308 - November 2009 Processors PowerQUICC II Pro MPC8306 - March 2011 Processors PowerQUICC II Pro MPC8309 - March 2011 Processors PowerQUICC III MPC8540 - June 2000 Processors PowerQUICC III MPC8541 - July 2001 Processors PowerQUICC III MPC8555 - July 2001 Processors PowerQUICC III MPC8560 - July 2002 Processors PowerQUICC III MPC8543 - September 2004 Processors PowerQUICC III MPC8545 - September 2004 Processors PowerQUICC III MPC8547 - September 2004 Processors PowerQUICC III MPC8548 - September 2004 Processors PowerQUICC III MPC8544 - September 2006 Processors PowerQUICC III MPC8572E - June 2007 Processors PowerQUICC III MPC8610 - September 2007 Processors PowerQUICC III MPC8536 - September 2008 Processors PowerQUICC III MPC8569 - February 2009 Processors 32-bit ColdFire - MCF5251 February 2007 Processors 32-bit ColdFire MCF51JU - March 2012 Processors 32-bit ColdFire MCF51JF - March 2012 Processors 32-bit ColdFire MCF51QU - March 2012 Processors 32-bit ColdFire MCF51QM - March 2012 Auto MCU 16-bit S12 - S12G October 2010 Auto MCU 16-bit S12 - S12HYx October 2009 Auto MCU 16-bit S12 - S12Px July 2009 Auto MCU 16-bit S12 MagniV - S12VR64 December 2011 Auto MCU 16-bit S12X - S12V December 2011 Auto MCU 16-bit S12X - S12XE September 2009 Auto MCU 16-bit S12X - S12XF March 2009 Auto MCU 16-bit S12X - S12XHY December 2010 Auto MCU 16-bit S12X - S12XS September 2009 Auto MCU 16-bit S12 MagniV - S12ZVL January 2015 Auto MCU 16-bit S12 MagniV - S12ZVM March 2014 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5554 July 2004 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5200 March 2005 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5534 June 2005 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5565 February 2006 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5566 May 2006 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5567 December 2006 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5200B September 2007 Auto MCU MPC5xxx 32-bit Power Architecture - MPC567XF October 2008 Auto MCU MPC5xxx 32-bit Power Architecture - MPC564xB-C October 2011 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5676R October 2011 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5777M December 2014 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5121e January 2009 Auto MCU MPC5xxx 32-bit Power Architecture - MPC563XM January 2009 Auto MCU MPC5xxx 32-bit Power Architecture - MPC551X September 2009 Auto MCU MPC5xxx 32-bit Power Architecture - MPC560XB December 2009 Auto MCU MPC5xxx 32-bit Power Architecture - MPC560xP April 2010 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5668G/E September 2010 Auto MCU MPC5xxx 32-bit Power Architecture - MPC567xK December 2012 Auto MCU 8-bit S08 - S08AWx January 2009 Auto MCU 8-bit S08 - S08LG32 March 2009 Auto MCU 8-bit S08 - S08SGx April 2009 Auto MCU 8-bit S08 - S08ELx September 2009 Auto MCU 8-bit S08 - S08SLx September 2009 Auto MCU 8-bit S08 - S08D November 2009 Auto MCU 8-bit S08 - S08MP16 November 2009 Auto MCU Kinetis EA - EA September 2014 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5553 July 2004 Auto MCU MPC5xxx 32-bit Power Architecture - MPC560xP April 2010 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5645S October 2012 Auto MCU MPC5xxx 32-bit Power Architecture - MPC564xA May 2012 Auto MCU MPC5xxx 32-bit Power Architecture - MPC564xL October 2009 Auto MCU MPC5xxx 32-bit Power Architecture - MPC564xS May 2012 Auto MCU MPC5xxx 32-bit Power Architecture - MPC5668G July 2012 Auto MCU MPC5xxx 32-bit Power Architecture - MPC567xK December 2012 DSP 24-bit DSP DSP56311 - October 2004 DSP 24-bit DSP DSP56321 - February 2005 DSP 24-bit DSP DSP56720 - May 2008 DSP 24-bit DSP DSP56721 - May 2008 DSP 24-bit DSP DSP56724 - August 2008 DSP 24-bit DSP DSP56725 - August 2008 DSP StarCore DSPs MSC8122 - May 2005 DSP StarCore DSPs MSC8156 - November 2008 DSP StarCore DSPs MSC8154 - April 2010 DSP StarCore DSPs MSC8252 - April 2010 DSP StarCore DSPs MSC8254 - April 2010 DSP StarCore DSPs MSC8256 - April 2010 DSP StarCore DSPs MSC8251 - April 2010 DSP StarCore DSPs MSC8151 - June 2010 DSP StarCore DSPs MSC8152 - June 2010 Sensors Accelerometer - MMA3201 January 2000 Sensors Accelerometer - MMA3202 January 2000 Sensors Accelerometer - MMA3204 January 2000 Sensors Accelerometer - MMA2201 May 2000 Sensors Accelerometer - MMA2202 May 2000 Sensors Accelerometer - MMA2204 May 2000 Sensors Accelerometer - MMA2301 May 2000 Sensors Accelerometer - MMA1254 May 2002 Sensors Accelerometer - MMA1260 November 2003 Sensors Accelerometer - MMA1270 November 2003 Sensors Accelerometer - MMA62xx June 2006 Sensors Accelerometer - MMA65xxKW November 2011 Sensors Accelerometer MMA766X - April 2009 Sensors Accelerometer MMA8451Q - February 2010 Sensors Accelerometer MMA8452Q - February 2010 Sensors Accelerometer MMA8491Q - February 2011 Sensors Touch Sensors MPR031 - February 2009 Sensors Touch Sensors MPR121 - December 2009 Analog Audio SGTL5000 - June 2008 Analog Battery Sensor - MM912_637 February 2011 Analog Communication Transceiver - MC33660 October 2011 Analog Embedded Microcontrollers + power - MM908E626 September 2004 Analog Embedded Microcontrollers + power - MM908E624 November 2004 Analog Embedded Microcontrollers + power - MM908E622 September 2005 Analog Embedded Microcontrollers + power - MM912_634 September 2010 Analog Embedded Microcontrollers + power - MM912F634 October 2010 Analog eXtreme Switch - MC33981 (MC12XS2) August 2004 Analog eXtreme Switch - MC33982 (MC12XS2) July 2006 Analog eXtreme Switch - MC33984 (MC12XS2) July 2006 Analog eXtreme Switch - MC10XS3412 October 2008 Analog eXtreme Switch - MC10XS3435 October 2008 Analog eXtreme Switch - MC15XS3400 October 2008 Analog eXtreme Switch - MC35XS3400 October 2008 Analog eXtreme Switch - MC33988 (MC12XS2) August 2010 Analog eXtreme Switch - MC10XS3535 August 2010 Analog eXtreme Switch - MC35XS3500 August 2010 Analog eXtreme Switch - MC09XS3400 March 2012 Analog eXtreme Switch - MC06XS3517 March 2012 Analog eXtreme Switch - MC10XS4200 (MC24XS4) June 2012 Analog eXtreme Switch - MC20XS4200 (MC24XS4) June 2012 Analog eXtreme Switch - MC06XS4200 (MC24XS4) September 2012 Analog H-bridge - MC33887 September 2006 Analog H-bridge - MC33932 January 2009 Analog H-bridge - MC33931 March 2009 Analog H-bridge MC34931 - January 2015 Analog H-bridge MC34932 - January 2015 Analog H-bridge - MC33926 May 2009 Analog H-bridge MC34933 - July 2010 Analog L/HS Switch - MC33879 December 2006 Analog LED Driver MC34844 - October 2008 Analog LED Driver MC34845 - June 2009 Analog LED Driver MC34848 - June 2010 Analog LIN Physical Layer - MC33662 September 2011 Analog LIN physical Layer - MC33663 August 2012 Analog Linear Regulator - MC33730 March 2009 Analog Power Management and user interface MC13783 - April 2006 Analog Power Management and user interface MC13892 - April 2010 Analog Power Management and user interface MC34708 - November 2011 Analog Power Management and user interface - MC34VR500 October 2014 Analog Power Management and user interface MMPF0100 MMPF0100 (Auto) October 2012 Analog Power Management and user interface MMPF0200 MMPF0200 (Auto) February 2014 Analog Powertrain and Engine Control - MC33810 July 2007 Analog Powertrain and Engine Control - MC33800 July 2007 Analog Powertrain and Engine Control - MC33811 July 2008 Analog Powertrain and Engine Control - MC33812 August 2009 Analog Powertrain and Engine Control - MM912_P812 July 2012 Analog Powertrain and Engine Control - MC33816 April 2014 Analog Pre-Drivers - MC33937 June 2008 Analog SBC - MC33989 November 2002 Analog SBC - MC33742 January 2004 Analog SBC - MC33742 June 2008 Analog SBC - MC33910 March 2009 Analog SBC - MC33911 March 2009 Analog SBC - MC33912 March 2009 Analog SBC - MC33904 November 2009 Analog SBC - MC33905 November 2009 Analog SBC - MC33903 March 2011 Analog SBC - MC33907 April 2014 Analog SBC - MC33908 April 2014 Analog Switch Detection Interface - MC33978 January 2015 Analog Switch Detection Interface MC34978 - February 2015 Analog Signal Conditioning - MC33975 February 2005 Analog Signal Conditioning - MC33972 July 2006 Analog Switching Regulator MC34712 - April 2007 Analog Switching Regulator MC34713 - April 2007 Analog Switching Regulator MC34716 - April 2007 Analog Switching Regulator MC34717 - April 2007 Analog Switching Regulator MC34704 - March 2008 Analog Switching Regulator MC34700 - April 2008 RF Land Mobile - AFT09MS031N April 2012 RF Land Mobile - AFT05MS031N April 2012 RF Land Mobile - AFT05MP075N January 2013 RF Land Mobile - AFT09MS007N June 2013 RF Land Mobile - AFT09MS015N December 2013 RF Land Mobile - AFT09MP055N July 2013 RF Land mobile - AFT05MS004N June 2014 RF RF Military MMRF1004N - December 2013 RF RF Military MMRF1005H - December 2013 RF RF Military MMRF1006H - December 2013 RF RF Military MMRF1007H - December 2013 RF RF Military MMRF1008H - December 2013 RF RF Military MMRF1009H - December 2013 RF RF Military MMRF1012N - February 2014 RF RF Military MMRF1014N - March 2014 RF RF Military MMRF1015N - March 2014 RF RF Military MMRF1016H - February 2014 RF RF Military MMRF1019N - March 2014 RF RF Military MMRF2004NB - December 2013 RF RF Military MMRF2005N - October 2014 RF RF Military MMRF2006N - April 2014 RF RF Military MMRF2007N - October 2014 RF RF Military - MMRF2010N June 2015 RF RF Military - MMRF5014H December 2014 RF RF Military - MMRF1020-04NR3 January 2014 RF RF Military - MMRF1304N December 2013 RF RF Military - MMRF1304L December 2013 RF RF Military - MMRF1305H December 2013 RF RF Military - MMRF1306H December 2013 RF RF Military - MMRF1308H March 2014 RF RF Military - MMRF1310H March 2014 RF RF Military - MMRF1315N June 2014 RF RF Military MMRF1316N - June 2014 RF RF Military - MMRF1318N June 2014 RF RF Military - MMRF1320N March 2015 RF RF Power Products - AFIC10275N November 2014 RF RF Power Products - MRF6V2150N February 2007 RF RF Power Products MRF6VP41KH - December 2007 RF RF Power Products - MRF6V14300H September 2008 RF RF Power Products - MRF6V12250H May 2009 RF RF Power Products - MRF6V12500H August 2009 RF RF Power Products MRF6VP121KH - December 2009 RF RF Power Products - MRFE6VP6300H October 2010 RF RF Power Products - MRFE6VP61K25H November 2010 RF RF Power Products - MRFE6VP5600H December 2010 RF RF Power Products - MRF8P29300H February 2011 RF RF Power Products - MRFE6VP8600H September 2011 RF RF Power Products - MRFE6VP100H April 2012 RF RF Power Products - MRFE6VS25N May 2012 RF RF Power Products - MRFE6S9060N/GN March 2007 RF RF Power Products - MRFE6VP5150N June 2014 RF RF Power Products - MRFE6VP5300N June 2014 RF RF Power Products - MRFE6VP61K25N February 2015 RF RF Power Products - MRF8VP13350N May 2015 RF RF Power Products - MRF7S24250N September 2015 Analog Power Management and user interface PF3000 - Jun 2015

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Product Longevity|Freescale

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Princeton Longevity Center Review – Health care center …

Monday, October 5th, 2015

Princeton Longevity Center Information

Princeton Longevity Center helps you prevents cancers and heart attacks, live longer, look and feel younger. Medicine has changed dramatically in the last forty years. Therefore, why are you still relying on your father's annual physical to stay well.

Services of Princeton Longevity Center

Our healthcare center provides state-of-the-art diagnostic facilities and a level of personal time, care and attention that is unmatched. Our healthcare center also offer a wide array of programs designed to meet the unique needs of busy corporate executives.

Our healthcare center equipped modern equipment such as 4-Slice CT Heart Scans and 3-D Virtual Colonoscopy our exams are able to detect the earliest evidence of the leading causes of death and disability years before any other tests. This will help our healthcare center to identify early and find appropriate treatment for you.

In addition, the best of all, while the Princeton Longevity Center's Executive Exams are the most comprehensive and effective you can find, we are also the most cost-effective. We commit that you will satisfied about cost and service while treatment

Contact Information

Center Name: Princeton Longevity Address: 136 Main Street, Forrestal Village Princeton, New Jersey 08540 Phone Number: 888-8000-PLC (toll-free) or (888.800.0752) Website: http://www.theplc.net Working Hours: For more information call (888.800.0752) or fax (609)430-8470

If you are unsatisfied about this center you can refer toHealth Care District of Palm Beach County. Read moreHealth Care District of Palm Beach County Reviews here.

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Longevity for You

Monday, October 5th, 2015

A holistic approach to autoimmune diseases! Click here for more information.

Stephen Lau

For more information, click here.

Longevity is the quest of many individuals. To live a long life and yet remain physically, mentally, and spiritually healthy is a challenge requiring great wisdom.

A long lifespan is a blessing, which has to be earned with the wisdom of an anti-aging healthy lifestyle.

Your Creator has given you the innate wisdom to live long, and your mission in life is to earn those extra years.

The objective of this site is two-fold:

To share with you my own experience of an autoimmune disease, and my self-awakening from that experience.

To share with you my search for health information on natural healing and

My mission is to provide you with

Your mission is to earn those extra years through a healthy longevity mindset to live an anti-aging and lifestyle. Longevity is for you, if, indeed, it is your passionate quest in life.

This is not a mission impossible: if there is a will, there must be a way.

You will be imbued with passion to live a longer tomorrow by changing not only your attitudes but also your lifestyle through a wealth of health information to accomplish your mission to earn your extra years.

Yes, the road to longevity may be long and winding, and even bumpy at times. But you need not fasten your seat-belt to the past or to the present (whatever the state of health you may be currently in). Rest assured, this site will continue to update you with new health information needed to earn your extra years. At the end of t-he day, you will be proud of yourself that you have a credential of living long that few would even dare to contemplate.

Stephen Lau

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Why Study Centenarians? An Overview New England …

Monday, September 21st, 2015

THE NEW ENGLAND CENTENARIAN STUDY

A Model of Aging Well. Centenarians (age 100+ years) markedly delay disability towards the end of their very long lives, at an average age of ~93 years (thats 33 years beyond the age of 60!).1 Thus, we regard these individuals as wonderful models of aging well. Some of our subjects, ~15% have no clinically demonstrable disease at age 100 years and we call them escapers. About 43% are delayers, or subjects who did not exhibt an age related disease until age 80 years or later. Finally, there are about 42% of our subjects who are survivors, or those with clinically demonstrable disease(s) prior to the age of 80 years.2 Supporting the compression of morbidity hypothesis, that as one approaches the limits of lifespan, diseases (morbidity) must be delayed (or escaped) towards the end of these longest lived, we have observed amongst supercentenarians (age 110+ years), that health span equals lifespan. Thus we believe that instead of the aging myth the older you get the sicker you get, it is much more the case of the older you get, the healthier youve been.

History The study began in 1995 as a population-based study of all centenarians living within 8 towns in the Boston area. The prevalence of Alzheimers Disease and other dementias in centenarians was the focus. At the time, the prevalence of centenarians in industrialized countries wasapproximately one centenarian per 10,000 people in the population. Thus, at anyone time, we were studying approximately 46 centenarians within a total population of 460,000 people (2). The NECS has gone on to enroll centenarians from throughout the United States and other countries and has grown to be the largest comprehensive study of centenarians in the world. There are currently ~1,600 centenarians, 500 children (in their 70s and 80s) and 300 younger controls. Amongst this group is the largest sample in the world, by far, of supercentenarians (age 110+ years) -there are about 107 of these oldest of the old subjects in our study.

Current and Previous Funders: We are tremendously thankful and beholden to the following foundations for supporting our enrollment and data collection efforts: the Alzheimers Association, the Ellison Medical Foundation, the Institute for the Study of Aging (now the Alzheimers Drug Discovery Foundation), the American Federation Aging Research, and the Glenn Foundation for Medical Research.Currently we receive our fundingfrom the National Institute of Aging (NIA), an institute of the National Institutes of Health (NIH), the William Wood Foundation and the Martin Samowitz Foundation. Our studies are supervised and approved by the Boston University MedicalCampus Institutional Review Board.

Demographics: In the U.S. and other industrialized nations, centenarians occur at a prevalence rate of about 1 per 6,000. When the centenarian study began in 1994, the prevalence rate was one per 10,000, making centenarians one of, if not the fastest growing segments of the population. In 2010, there are about 80,000 centenarians in the U.S.

Eighty-five percent of cenenarians are women and 15% are men. Among supercentenarians, the female prevalence may increase to about 90%. Though women by far and away win the longevity marathon, paradoxically the fewer men are generally functionally better off and healthier. This may be because women handle age related diseases better (how they do this is not clear) whereas at these ages, the men more readily die from them. Thus, the men who survive have to be relatively healthy and functionally fit.

Supercentenarians, people who are 110+ years old occur at a rate of about 1 per 7 million. In 2010, there are about 60 to 70 supercentenarians in the US. In June, 2010, the New England Centenarian Study enrolled its 107th supercentenarian, thus constituting by far and away the largest sample of such subjects in the world.

Geography: There are several geographical areas that have claimed inhabitants with extreme longevity, but after closer examination, these claims have been found to be false. Vilacamba, Ecuador almost became a tourist attraction because natives claimed their water was a fountain of youth leading to the many super-centenarians (age >110 years) in that region. What about the reports of people in the Russian Caucases living to 150 years and beyond? Remember the Dannon yogurt commercials? In fact, those purported super-centenarians were taking on the identities of their parents, aunts and uncles. The oldest person from whom we have multiple forms of proof-of-age is Madame Calment who died at the age of 122 years in 1997.

These regions of purported exceptional longevity still merit careful study however. Though claims of extreme age are untrue, there still may be an unusually high prevalence of very old fit people in these regions. In the Tibetan mountains for instance, octogenarian and nonagenarian elders, impressively many of them men, still herd live stock and still lead physically strenuous lives.

Predictors of Reaching 100: Once it truly became apparent that living to 100 was a terrific advantage, not just in years of survival but importantly in many more years of quality life, we set out to understand what factors the centenarians had in common that might explain such an advantage. Not all centenarians are alike. They vary widely in years of education (no years to post-graduate), socioeconomic status (very poor to very rich), religion, ethnicity and patterns of diet (strictly vegetarian to extremely rich in saturated fats). However, the centenarians we have studied do have a number of characteristics in common:

Nature Versus Nurture: The Role of Genes Versus Environment in Aging and Exceptional Longevity. Gerontologists often cite studies of lifespans amongst identical twins reared apart to describe the genetic and environmental components of aging. Based upon these studies, the common answer is 70-80%environment and 30-20% genes. This makes sense in the context of results from the study of Seventh Day Adventists at Loma Linda University who as a group have perhaps the longest average life expectancy in the United States, 88 years or 88 years for men and 89 years for women. The main attributes that these individuals have in common is that their religion for the most part asks that they have very good lifestyle choices. That is, they tend to be vegetarian, they dont smoke, they regularly exercise and hey spend a lot of time with their families and with their religion. Many Americans do the opposite (e.g. excessive meat consumption, lack of exercise, smoking, etc) and thus it is not surprising that on average, Americans die 8-10 years sooner. What the 7th Day Adventist results also show us is that the average American has the genes to reach their mid-late 80s, they just nee to take very good care of themselves with proper lifestyle choices. Also note that the oldest subects in the twin studies lived to their early to mid-eighties. Therefore, again, these findings indicate what it takes to live to what should be average life expectancy for most of us, age 86 years for men and 89 years for women.

However, we have learned from our studies of the siblings of centenarians and of supercentenarians that excepional longevity runs very strongly in families. Also, a Danish study of nonagenarians and centenarians has noted that the power of an exceptional longevity (EL)study to discover genetic factors associated with EL increases with the age of the subjects. These and other study results strongly suggest that the genetic component of exceptional longevity gets larger and larger with increasing age and is especially high for those age 106 years and older. The week of July 1, 2010, we will have a paper come out in Science that dilineates the roles of genes in exceptional longevity much more clearly (media embargoed until July 1, 2 pm).

Neuropsychological and Neuropathological Studies. We are particularly interested in how centenarians are able to markedly delay or in some cases escape Alzheimers disease. We perform detailed and annual neuropsychological examinations on centenarians living within 3-4 hours of Boston. A number of these subjects have indicated their willingness to donate their brains for neuropathological studies once they pass away, thus allowing our scientific collaborators to better characterize the health of the centenarians exceptionally old brains.

We also expend significant resources to disseminate our findings and to advocate for older people, providing an optimistic and enabling view of aging. Most people have the genetic makeup to live into their mid to late eighties in very good health, and like centenarians, compress the time they are sick towards the end of their lives. Much of their ability to do so depends upon healthy behaviors including a diet conducive to being lean, not smoking, and strength training exercise. Promoting this philosophy will have a much greater impact now on many more people than our genetic research. Our collaborations with Dr. Robert Butler and the International Longevity Center have been extremely fruitful in furthing this mission.11-13

The New England Centenarian Study Boston Medical Center 88 East Newton Street, B-2400 Boston, MA 02118 Local Phone: (617) 638-6679 or (617) 638-6688

Toll Free: 1-888-333-6327

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Longevity Diet: Healthy Anti-aging Foods – WebMD

Wednesday, September 9th, 2015

Is your diet the key to longevity? Find out why eating right just may mean aging right, too.

Aging: everyone does it, yet some people seem relatively unaffected by getting older. Could good nutrition be the key to a healthier, longer life?

Does Aging Equal Illness?

The Secret to Healthy Aging

Who doesn't wish for a fountain of youth? Magical youth-restoring springs exist only in legend, but science does point to a few simple, healthy habits that can help extend your life. Heather Whitson, MD, associate professor of medicine (geriatrics) at Duke University School of Medicine, shares the most effective tips.

Read the The Secret to Healthy Aging article > >

"Aging is often associated with the development of one or more chronic diseases, but it doesn't have to be that way," says Jeffrey Blumberg, PhD, professor at the Friedman School of Nutrition Science and Policy at Tufts University.

It's not always just a matter of time before you have a heart attack or stroke, get type 2 diabetes or cancer, break a hip because of osteoporosis, or develop Alzheimer's, even though these conditions are often associated with aging, Blumberg says.

Your risk for disease and disability increases with inadequate physical activity, genetic susceptibility, and poor diet.

Aging: Defy It With Diet

So what's the best eating plan for preventing, delay, or minimizing the conditions associated with aging, including inflamed joints, flagging memory, and failing eyesight?

"The most beneficial diets rely heavily on fresh vegetables, fruits, and legumes -- foods that are naturally lower in calories and packed with nutrients," says Bradley Willcox, MD, MPH, co-author of The Okinawa Diet Plan and professor of geriatrics at the University of Hawaii.

Experts suspect the antioxidant compounds found in produce, legumes, and whole grains are largely responsible for holding back the march of time.

Antioxidants, such as vitamins C and E, and other compounds, including polyphenols and anthocyanins, battle free radicals -- unstable forms of oxygen that damage cell function. Free radicals form from normal metabolism. Your body also produces them in response to strong ultraviolet rays from the sun; air pollution; smoking; and secondhand smoke.

The buildup of free radicals contributes to the aging process and to the development of a number of age-related diseases such as cancer, heart disease, and inflammatory conditions, including osteoarthritis. What's worse, aging increases free radical production. That means your diet should be healthier than ever with the passage of time.

The question, of course, is how do we do that?

Anti-Aging Nutrition

Antioxidants generate a lot of buzz when it comes to longevity, but aging well takes more. You must optimize a myriad of beneficial nutrients, including protein, calcium, and vitamin D, and minimize detrimental dietary components including saturated and trans fats.

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Longevity – What is Longevity?

Wednesday, September 9th, 2015

Updated May 01, 2014.

Definition:

long life, a great duration of life, coming from the Latin longaevits. In this word, you can see the words longus (long) and aevum (age).

The most important part of this definition is the comparative nature of it. Long life implies longer than something - and that something is the average life. If you live longer than the average person, then you could be said to have longevity. Striving for you maximum potential age is the goal of longevity.

This can be reached through creating healthy behaviors and attitudes. Biologists sometimes define longevity as:

the average life span expected under ideal conditions.

So while the life expectancy of people in the US is somewhere around 78, our longevity might be much higher. We all might live longer if we can create the ideal conditions of a healthy diet and exercise.

You may think that your genes determine your longevity, but the truth is that genetics account for a maximum of 30 percent of your life expectancy - the rest comes from your behaviors, attitudes, environment and a little bit of luck.

You may have heard about various life extension techniques. Keep in mind that none of them have been proven in humans and most are just theories. The only proven way to live longer is to live healthy. So make a commitment today to make a one healthy change a week. Before you know youll be feeling better and on the road to longevity.

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Longevity – How To Live Longer – Secrets to Longevity

Wednesday, September 9th, 2015

1. Stand up while reading articles like this one ...and (especially) while watching TV. Australian researchers determined that every hour of couch-potato-ness docks 21.8 minutes from a person's life.

2. Join a book club. People with a solid group of friends are 50 percent more likely to survive at any given time than those without one, found Carlin Flora while researching her book Friendfluence. Researchers from Brigham Young University calculated that being a loner is an equivalent mortality risk to smoking 15 cigarettes a day, says Floraeven riskier than being obese or not exercising.

3. Better yet, join a French Words et Wine or Mandarin for Beginners group. Canadian scientists used CT scans to compare the brains of bilingual and monolingual Alzheimer's patients and found that being able to speak multiple languages seemed to keep patients cognitively agile for longer. Protective benefits start in childhood, but the research suggests that picking up a new language later in life may also help stave off dementia.

4. Nurture your java habit... Drinking four cups of brewed coffee (or the amount of caffeine that you'd get in one Starbucks venti) a day has been linked to as much as a 50 percent lower risk of developing type 2 diabetes, a 25 percent lower risk of endometrial cancer, and a 20 percent lower risk for depression. (Keep in mind that this daily amount may cause those who are new to the coffee habit to feel jittery and have trouble sleeping.)

5. ...or fill your mug with tea. In a study of more than 40,500 Japanese men and women, those who drank five or more cups of green tea every day had the lowest risk of dying from heart disease and stroke. Other studies involving black tea showed similar resultsbut adding milk may cancel out tea's cardiovascular benefits. Whatever color tea you choose, drink it black, or with honey and lemon.

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Amazon.com: Customer Reviews: LONGEVITY Migweld 140 – 140 Amp …

Wednesday, September 9th, 2015

Only took a few minutes to get the welder out of the box, attach the torch, and load a roll of flux core wire (Note that you have to buy your own flux core; it comes with solid wire). Only took three tries to guess a voltage/feed rate combination to get a decent weld; I'm sure that as I become more familiar with the unit (and/or read the instructions) I'll get more reliable.

I ended up buying from Longevity directly: it's a 1 year warranty through Amazon but a 5 year warranty from the factory. Shipping was quite a bit slower and was not free, but they worked with me on the price and came in around what the Amazon cost was.

In terms of quality, the ground clamp is a bit weaker than I'm used to and is more slippery, but the unit seems generally solid where it matters. It's not as nice as my Dad's high-end digital Lincoln, but it's also a heck of a lot cheaper and more portable. I haven't used it much, so I'm not sure about durability yet, but I have a good feeling about it. It feels well constructed.

Regarding the price and features, I did a LOT of shopping around and at under $400 (total cost of the unit, shipping, and taxes) the features of this welder seem to be either comparable to or better than welders in the $550-$600 range (such as the Hobart Handler and the Lincoln Easy Mig 140). I've also used cheaper ~$100 Harbor Freight 110V welders, and this was FAR better: the tip of this welder is only hot when the trigger on the torch is pulled, the penetration was much better, and the build quality felt better.

Regarding customer service, I'm very impressed. I mistakenly thought that the argon regulator was missing from the box, called customer service, and they promptly, with absolutely no arguing, put in a warranty order to ship me a replacement. After I found the regulator, they were very polite when I called back to own up to my mistake.

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Longevity myths – Wikipedia, the free encyclopedia

Wednesday, September 9th, 2015

Longevity myths are traditions about long-lived people (generally supercentenarians), either as individuals or groups of people, and practices that have been believed to confer longevity, but for which scientific evidence does not support the ages claimed or the reasons for the claims.[1][2] While literal interpretations of such myths may appear to indicate extraordinarily long life spans many scholars[3] believe such figures may be the result of incorrect translation of numbering systems through various languages coupled by the cultural and or symbolic significance of certain numbers.

The phrase "longevity tradition" may include "purifications, rituals, longevity practices, meditations, and alchemy"[4] that have been believed to confer greater human longevity, especially in Chinese culture.[1][2]

Modern science indicates various ways in which genetics, diet, and lifestyle affect human longevity. It also allows us to determine the age of human remains with a fair degree of precision.

Age claims for the earliest eight Sumerian kings in the major recension of the Sumerian King List were in units and fractions of shar (3,600 years) and totaled 67 shar or 241,200 years.[5]

In the only ten-king tablet recension of this list three kings (Alalngar, [...]kidunnu, and En-men-dur-ana) are recorded as having reigned 72,000 years each.[6][7] The major recension assigns 43,200 years to the reign of En-men-lu-ana, and 36,000 years each to those of Alalngar and Dumuzid.[5]

In the Hebrew Bible, the Torah, Joshua, Job, and 2 Chronicles claim several individuals with long lifespans. Students of the Bible hold various positions regarding the ages given in the Bible. Some assert a literal translation while others search for a less dogmatic interpretation. ". . . patient research has gone a long way towards resolving this knotty problem. [8]

Some literary critics explain these extreme ages as ancient mistranslations that converted the word "month" to "year", mistaking lunar cycles for solar ones: this would turn an age of 969 "years" into a more reasonable 969 lunar months, or 78 years of the Metonic cycle.[9]

Donald Etz theorized that the Genesis 5 numbers were multiplied by ten by a later editor.[10] A similar scenario is believed to have led to some confusion as the mystery of Plato's Atlantis.[11] Critics, however, believe this would be inconsistent as the ages of the first nine patriarchs at fatherhood, ranging from 62 to 230 years in the manuscripts, would then be transformed into an implausible range such as 5 to 18 years.[12]

Numbers from the ancient near east are recognized as inflated through some source of number manipulation complex patterns are suggested. The Sumerian tradition suggests certain kings reigned for 36,000 years.[13] This makes the current numbers in Genesis for the antediluvians seem extremely conservative. The number of years each Sumerian king reigned, as Dwight Young has pointed out, is often a square number or the sum of squares. For example, reigns of 900 years (302); 324 (182); 136 (102 + 62); and 116 (102 + 42) are recorded.[14] This ancient tradition of manipulating numbers can also be found in the ages the Old Testament assigns to the patriarchs. Abraham is reported to have lived, according to the Hebrew Bible (Leningrad Codex), to the age of 175. His son, Isaac, lived to be 180. Abraham's grandson, Jacob, lived only to the age of 147. And Joseph, Jacob's son, lived the shortest life of all110.[15] However, like the reigns of some of the kings in the Sumerian King List, the ages of the patriarchs are products of a multiplier and a square and in one case the sum of squares. There is a mathematical progression in the ages of the patriarchs.

This leaves students of the Bible to speculate the reasons why translators of the text would format numbers in such a manner. Several suggestions are below.

Others point out that Biblical genealogies contain generational gaps. For example Paul Y. Hoskisson pointed out that between Ozias and Joatham in verses 8 and 9, Matthew left out Joash, Amaziah, and Azariah (Joash was the son of Ozias [Ahaziah in 2 Kings 11:2] and the father of Amaziah, grandfather of Azariah and great grandfather of Joatham [Jotham in 2 Kings 15:7]). The Gospel of Luke more realistically has 56 ancestors from Abraham to Christ.[16] Hoskisson further suggested that the gematria of King David's name may have something to do with Matthew's choice of the number "fourteen." The Hebrew letters in David's name, , given their numerical value, add up to the number fourteen. Since the writer of the Gospel of Matthew divided the genealogy into three sections, each containing 14 generations in accordance with the numerical value of David's name; Abraham to David, David to the Exile, and the Exile to Christ[17] certain names would have been omitted which the author of Luke had access to. Nineteenth-century critic Vincent Goehlert suggests the lifetimes "represented epochs merely, to which were given the names of the personages especially prominent in such epochs, who, in consequence of their comparatively long lives were able to acquire an exalted influence."[18]

Biblical scholars that believe in literal translation give explanations for the advanced ages of the early patriarchs. In one view man was originally to have everlasting life, but as sin was introduced into the world by Adam and Eve, its influence became greater with each generation and God progressively shortened man's life. The Biblical upper limit of longevity was categorized by the Bible scholar Witness Lee as having four successive plateaus of 1,000, 500, 250, and finally 120 years,[19] and "four falls of mankind" correspond to these four plateaus.[20] In a second view, before Noah's flood, a "firmament" over the earth (Genesis 1:68) contributed to people's advanced ages.[21]

The reigns of several shahs in the Shahnameh, an epic poem by Ferdowsi, are given as longer than a century:

Lucian wrote about the "Seres" (a Chinese people), claiming they lived for over 300 years.

Some early emperors of Japan ruled for more than a century, according to the tradition documented in the Kojiki, viz., Emperor Jimmu and Emperor Kan.

In Roman times, Pliny wrote about longevity records from the census carried out in 74 AD under Vespasian. In one region of Italy many people allegedly lived past 100; four were said to be 130, others even older. The ancient Greek author Lucian is the presumed author of Macrobii (long-livers), a work devoted to longevity. Most of the examples Lucian gives are what would be regarded as normal long lifespans (80100 years).

In legend, Praotec ech ("forefather Czech", 342680) lived 338 years.[citation needed] And Pemysl, the Ploughman (founder of the Pemyslid dynasty) could have lived for more than 180 years (561745).[citation needed]

Social Security:

A 1973 National Geographic article on longevity reported, as a very aged people, the Burusho or Hunza people in the Hunza Valley of the mountains of Pakistan.[57]

Deaths officially reported in Russia in 1815 listed 1068 centenarians, including 246 supercentenarians (50 at age 120155 and one even older).[30]Time magazine considered that, by the Soviet Union, longevity had elevated to a state-supported "Methuselah cult".[58] The USSR insisted on its citizens' unrivaled longevity by claiming 592 people (224 male, 368 female) over age 120 in a 15 January 1959 census[59] and 100 citizens of Russia alone ages 120 to 156 in March 1960.[60] Such later claims were fostered by Georgian-born Joseph Stalin's apparent hope that he would live long past 70.[58]Zhores A. Medvedev, who demonstrated that all 500-plus claims failed birth-record validation and other tests,[58] said Stalin "liked the idea that [other] Georgians lived to be 100".[60]

Swedish death registers contain detailed information on thousands of centenarians going back to 1749; the maximum age at death reported between 1751 and 1800 was 127.[63]

Swiss anatomist Albrecht von Haller collected examples of 62 people ages 110120, 29 ages 120130, and 15 ages 130140.[65]

The idea that certain diets can lead to extraordinary longevity (ages beyond 130) is not new. In 1909, lie Metchnikoff believed that drinking goat's milk could confer extraordinary longevity. The Hunza diet, supposedly practiced in an area of northern India, has been claimed to give people the ability to live to 140 or more.[68] There has been no proof that any diet has led humans to live longer than the genetically-recognized maximum[citation needed] (currently the oldest verified person, Jeanne Calment, died at age 122.45 years),[69] however Caloric restriction diets have increased lifespans of rodents significantly.

Traditions that have been believed to confer greater human longevity include alchemy.[4]

The Fountain of Youth reputedly restores the youth of anyone who drinks of its waters. The New Testament, following older Jewish tradition, attributes healing to the Pool of Bethesda when the waters are "stirred" by an angel.[73]Herodotus attributes exceptional longevity to a fountain in the land of the Ethiopians.[74] The lore of the Alexander Romance and of Al-Khidr describes such a fountain, and stories about the philosopher's stone, universal panaceas, and the elixir of life are widespread.

After the death of Juan Ponce de Len, Gonzalo Fernndez de Oviedo y Valds wrote in Historia General y Natural de las Indias (1535) that Ponce de Len was looking for the waters of Bimini to cure his aging.[75]

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Home | Longevity

Wednesday, September 9th, 2015

Evidence-based Anti-Aging Medicine is rapidly becoming the future of medicine and can be easily integrated into your family or internal medical practice.

We provide a comprehensive certification program that will thoroughly prepare you for successful integration of Anti-Aging Medicine into your existing practice. Longevity is Lifes practical educational modules were developed from over 45 years of combined personal professional experience of our experts and from information presented at multiple Anti-Aging Congresses and Workshops in Europe and USA. They are designed to distill an inordinate amount of information into practical and easy to implement course on Anti-Aging Medicine. After completion of our course, you will return to your practice energized and equipped with new treatment strategies that will benefit both your practice and your patients.

With Longevity is Lifes Anti-Aging medical training you will be able to offer patients a personalized, healthy approach to looking and feeling great. Medical science has seen the most noticeable advancements in these areas, in recent years, and the clinical data is constantly being updated and yielding significant results. Our program is evidence-based, and thoroughly goes over the latest necessary standards of care, medical protocols and testing, so that you will be able to utilize and seamlessly integrate this field of medicine within your clinical practice.

Our modules are taught by world recognized physician expert in Anti-Aging medicine. Nataliya Chekalska, MD, PhD is a speaker and a former scientific coordination for many years of Anti-Aging Medicine and Aesthetic World Congress and has extensive experience in her field. She is the author of several patents of IV Anti-aging therapy protocols, which are now widely used throughout Europe and US.

Anti-Aging medicine is an extension of preventive health care. If you are testing your patients cholesterol, or prescribing melatonin or DHEA, or suggesting a mammogram, you are already involved in certain aspects of Anti-Aging medicine. It is estimated that more than 90% of all adult illness is due to the generative processes of aging, which may be corrected or improved by the addition of supplements, detoxification and hormonal correction.

Anti-Aging is a relatively new field of medicine, where natural approaches and lifestyle changes are used, in conjunction with traditional medicine, where applicable, to give relief to a variety of conditions. The total market within the United States, as it relates to Anti-Aging medicine, is estimated to be over $15B.

During the Anti-Aging training modules you will understand how to analyze, test, evaluate and treat numerous chronic and age-related conditions you are presented with, each day. Participants can pass our on-line certification tests and will be awarded Longevity is Life Certificate of Completion as a Preventive and Anti-aging Medicine Professional.

Throughout the aging process, our internal systems and body chemistry change, and we develop certain imbalances and deficiencies, which in the past we would explain away as caused by life changes and genetics. Today we are able to review these various primary and secondary hormones, through testing and systems reviews, and to develop an effective personalized treatment plan that reverses these chronic conditions.

As we get older, our metabolism gets less efficient, thereby the absorption of nutrients and the excretion of toxic cellular wastes become less effective. Age related nutritional deficiency diseases become more frequent and the effects of chronic stress and poor nutrition can contribute to the buildup of free radicals that in turn can lead to more and more symptoms of premature aging like loss of skin elasticity and other metabolic problems. Anti-aging therapies can offer the opportunity to help reduce oxidative stress, and supply the vital vitamins, minerals and nutrients that are needed to help repair cellular damage and restore homeostatic balances.

Even when a person thinks that he is eating well, he may not be assimilating all the proper nutrients he needs to stay at optimal health because the absorption of nutrients is a critical factor. Oral vitamins commonly have absorption rates of only 10 to 15 percent and decreases in the efficiency of the digestive tract as we are or disease can also reduce absorption efficiencies.

The importance of standing nutrition therapy programs after the age of 35 cannot be overemphasized. This is a best time to mitigate many of the age effects of free radicals, hormone depletion, glycation, membrane damage and toxin accumulation. After 35 years old natural human growth hormone begins to tape off and the efficient physiological processes that worked well in youth begin to become less efficient. At this age they need more nutrition and support to maintain our health.

Most of the age related diseases like heart disease, cancer and diabetes as well as a whole host of other deficiency related diseases begin to act on the general population in almost a logarithmic-like way after 50 years of life. The pace of aging increase as we age. At the age 50 there is 400 % increase in the incidence of age related diseases. This goes to a thousand fold increase by age 60 and peaks at 2500% increase by age 80.

Its the growing trend- customized IV cocktails providing in wellness centers and in all top quality spas worldwide. Iv therapy was introduced more than 50 years ago- now its a complimentary treatment IV therapy actually works at the cellular level.

To re-iterate, IV nutrition provides direct access to circulatory system bypassing any deficiencies in the normal gastrointestinal digestion and assimilation process. It is the route for drug or nutrient delivery that might normally irritate gastric mucosa or might not be properly integrated with any other type of delivery. IV administration is likewise an effective route for instant drug or component action that avoids having to wait to address critical problems. Additionally, it effortlessly allows for instant drug termination if sensitivity or adverse reactions occur.

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Dr Joel Wallach Colloidal Minerals Resource Center

Wednesday, September 9th, 2015

A colloid is a particle substance that retains its identity and remains in liquid suspension. Colloids are very small in size and therefore easily absorbed by the cells of the body. Plants convert metallic minerals into this form.

Dr. Carey Reams, a well known biophysicist and biochemist, discovered that colloids can get so small they can go through glass. And that even within a colloid there can be a "solar system" of different minerals. They have a natural negative electrical charge.

Dr. Reams found that the human body requires 84 of the known 106 elements to maintain optimum health. Many more than is currently recognized.

In 1925, a historic discovery was made by a rancher in Central Utah. Through an unusual set of circumstances the mineral remains of an ancient rain forest, determined by the US. Dept. of Interior to be from the Cretaceous Period, approximately 60 million to 127 million years old, were encapsulated and preserved.

The material is humic shale, not bentonite, clay or dried up sea beds which is the most common source of so called Colloidal Minerals.

Only a small portion from various parts of the deposit are used to insure a balance of the over 65 various minerals and trace minerals that form the exclusive Majestic Earth product that meets Dr. Wallach's quality standards, the rest is sold to bulk processors and distributed under various no name labels.

Only the Majestic Earth products represents the true plant derived product. Time tested by 70 years of use. This is the product all others try to compare themselves with.

These rare colloidal plant minerals are extracted with pure, cool clear water to the desired concentration and then processed through a complex array of special filters to allow only the organic colloidal minerals in the final solution. There are no preservatives, flavorings. or synthetic substances added.

Dr. Wallach's Majestic Earth liquid minerals have a natural negative electrical charge, which is the signature of the true plant derived mineral. This has two very important benefits.

One, it greatly increases the transport and bioavailability of other nutrients gotten from foods and/or vitamins and other supplements.

And two, it will attract toxins and heavy metals from the body and flush them out. Clinical tests done here in the US. and in Germany have verified this.

The mineral product that Dr. Joel Wallach is referring to on his audio tape, "Dead Doctors Don't Lie", is a plant derived product which has been on the market for 70 years.

Dr. Wallach exclusively represents The Youngevity Family of Companies" and only endorses or recommends their plant derived liquid colloidal mineral products.

Minerals in their metallic form can be toxic to the human body. Dangerous levels of these metallic minerals can accumulate in the tissues and create serious health problems.

These are found in the 2000 year old sea bed deposits, and are loaded with chloride. Very toxic inorganic minerals are being sold to an unsuspecting public under the guise of colloidal. The key factor is true plant derivation.

Plant source minerals, produced for Dr Wallach and Youngevity are completely non-toxic, Plant Derived, and is proven by its 70 year history of positive results with the thousands of its customers, and verified by the following scientific test done in Germany.

Dr Joel Wallach Youngevity Vitamin and Minerals

Dr. Fritz-Albert Popp, a biophysicist and professor at the Kaiserlautern University in Germany, developed a very sophisticated and precise method of determining whether or not a substance is toxic or to what degree it is beneficial to live cells.

This method accurately measures the actual life energy output of these cells. The minerals not only tested completely non-toxic, but increased the cell's life energy output beyond what Dr. Popp had established as his optimum output benchmark.

He congratulated us on the excellence of the product. No other manufacturer of natural colloidal minerals has this test guaranteeing the safety and effectiveness of their product.

EDITORS NOTE: As with any change in your diet or nutrition, see your health practitioner about utilizing this information. This information in no way attempts to construe a diagnosis, cure or treatment. **These statements have not been evaluated by the FDA. For any claims linking health to nutrition that is not "classic".

Sincerely, Joel D. Wallach, B.S., DVM, N.D.

Dr Joel Wallach Youngevity Vitamin and Minerals

than all the wars in American history. . .

You get all the nutrition you need from the four food groups."

But those raw materials so vital to your health can be hard to find. Centuries of mining, farming, irrigation, and acid rain have eroded life-giving minerals from our soil. These mineral-deficient soils are then used to grow and cultivate our food. Because of this, our food may be lacking in essential minerals

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longevity | Chiraan’s Astrology

Wednesday, September 2nd, 2015

AyuhKshinam Ayuhvriddhi

When 8th and ninth lord in 12th then one will lose his longevity by the dishonouring the elders. Aspect of benefics on 8th house and its lord increases the longevity.Having good Ayur yogas also increases the strata of longevity .12th sun for libra ascendant also confers longevity.

One loses his longevity by

Once there was a very learned bramhin by name Srinivasachar in 18th century. He was a great scholar in sanskrit and philososphy . He was very proud of his scholarly aptitude . It was also around same time ,when bhakti movement had gained importance in south India and Das culture was on the rise . Das way of life was initiated by Purandardas [incarnation of Narada] in the south when Vijaynagar empire was at its peak under the able guidance of Vyasateertha [Incarnation of Balhik] a pontiff in the line of Madhvacharya. Das culture mainly emphasised the delivery of Vedic Knowledge in local language [Kannada] through poems and melodious songs for the Lord Vittala .Thus common men and women were suddenly bestowed the nectar of geeta and puranas in the common mans language then prevalent. Vijaydas [ incarnation of sage BHrigu] was also one such illustrious personality of those times. VijayDas was also maternal uncle to Srinivasachar.

Srinivasachar owing to the pride ,he had cultivated, did not recognize the fact that philososphy could be delivered to common man in simple mans language. The philosophy which could not be guaged by immense learning in sanskrit ,how could they be delivered in simple dismal languages.

True ! Sanskrit is a complex language ; its grammar is vast ; its difficult to master it ;perhaps takes many years for the same; Then comes the grasp poetry in sanskrit ! still difficult ; upon that philosophy ; its totally incomprehensible ; Almost all the philosophic literature is available in archaic sanskrit in poetry form ; difficult to decode. it takes many years of study and mastery to exhibit command over such philosophic understandings. All such efforts would seem ridiculous , if somebody claims he understands philosophy although he does not understand sanskrit. Amazing still will be the statement if one says he can express all the intricacies of the Vedas in common mans language! even if we were to accept this ; How come the common man is going to understand them; Can such claim be accepted ?

SrinivasAchar felt these Das culture guys were fooling people in the name of God for common man and dancing around with bells in their anklets;He seriously doubted their credentials and claims of sainthood.

One day VijayDas came visiting to his place and even visited his house. Srinivasachar out of ego and pride did not respect the Learned VijayDas and did not even welcome him in his house. VijayDas went away smiling .But soon after this insult , Srinivasachar started losing out on health.Slowly his health deteriorated. He contracted Kushtha [skin disease ] and slowly his moments became restricted.

SrinivasAchar now contemplated he is heading for Apamrutyu [ his longevity has decreased ]. He however could not comprehend the loss of longevity[ Ayuhkshina] . So to gain longevity he prayed Lord Hanuman in the mountains near Panchamukhi on the banks of TungaBhadra river. After 42 days of Vayustuti Purascharan ,he had a dream , where Lord Hanuman directed him to pray Shri Raghavendra swamy in Mantralaya for further directions. [ Shri Raghavendra has been specially sent from heavens by God to grant the wishes of the humans, hence the direction ].

Srinivas achar spent next few days under austere conditions as penace towards Shri Raghavendra Swamy at Mantralayam. The following night Shri Raghavendra swamy appeared and explained to him the reason for his loss of longevity .[ Disrespect towards very learned VijayDas] ,so he asked him to seek his pardon and blessings.

Srinivas Achar went to VijayDas and sought pardon and accepted the supremacy of poetry and dance in gaining lords Grace . VijayDas directed him to go to his disciple Shri GopalDas [incarnation of Ganapathy] to seek blessings and accept him as Guru .GopalDas was a very learned man , he kept the entire place clean of pebbles lest his disciple would be hurt [ Srinivasachar was troubled by lack of moment and kushtha ] .

When SrinivasAchar came and bowed to GopalDas , he transfered his 40 years of longevity [ through yoga] to a Roti of Jowar[Bhakri] and asked him to eat it to gain a 40 years. When one loses longevity owing to disrespect no austerities can actually increase it . someone has to donate his longevity to make a person live more. [SO never ever lose longevity we would not find anyone to donate life] Without life and longevity all the riches and fame are useless.

Despite donating 40 years GopalDas lived for 80 years of age and gave to this world a wonderful science and technique of Vishwopasana . He rechristened Srinivasachar as JagannathDas [ srinivasachar is incarnation of AHLAD brother of Prahlad , and hence had a very special relationship with Shri Raghavendra swamy ,a incarnation of Prahlad]

JagannathDas gave a magnum opus HARIKATHAMRUTASARA a treatise on the qualities of God Narayana in the comman mans language specially to be read by women and others inelligible for Vedas.

krishnarpanamastu

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longevity | Chiraan's Astrology

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